Giuseppe Chiantia

Università degli Studi di Torino

Italy

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Articles (12)

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2025

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2024

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2023

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2023

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2023

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2023

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2022

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2022

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2020

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2020

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2020

Collaborators (10)

Antonia Gurgone

University of Turin

ITALY

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Andrea Marcantoni

University of Turin

ITALY

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Alex Costa

Professor

University of Milan

ITALY

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Enis Hidisoglu

Asistant Professor

University of Turin

ITALY

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Tommaso Pizzorusso

Full Professor

Scuola Normale Superiore

ITALY

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Maurizio Giustetto

Associate Professor of Human Anatomy

Università degli Studi di Torino

ITALY

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Silvia De Rubeis

Associate Professor

Icahn School of Medicine at Mount Sinai

UNITED STATES

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Luca Palazzolo

Assistant Professor of Applied Physics

Università degli Studi di Torino

ITALY

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Emilio Carbone

Full professor, Deputy Director of Research

University of Turin

ITALY

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Valentina Carabelli

Full Professor in Physiology

University of Turin

ITALY

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Giuseppe Chiantia

Research Interests

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Articles (12)

Glyphosate impairs both structure and function of GABAergic synapses in hippocampal neurons

Preclinical alternative drug discovery programs for monogenic rare diseases. Should small molecules or gene therapy be used? The case of hereditary spastic paraplegias

Missense variants in RPH3A cause defects in excitatory synaptic function and are associated with a clinically variable neurodevelopmental disorder

The Role of Ryanodine Receptors in Regulating Neuronal Activity and Its Connection to the Development of Alzheimer’s Disease

Two firing modes and well-resolved Na<sup>+</sup>, K<sup>+</sup>, and Ca<sup>2+</sup> currents at the cell-microelectrode junction of spontaneously active rat chromaffin cell on MEAs

Frontal EEG alterations induced by hippocampal amyloid pathology in rats

mGluR5 PAMs rescue cortical and behavioural defects in a mouse model of CDKL5 deficiency disorder.

Loss of ARHGAP15 affects the directional control of migrating interneurons in the embryonic cortex and increases susceptibility to epilepsy

The ryanodine receptor-calstabin interaction stabilizer S107 protects hippocampal neurons from GABAergic synaptic alterations induced by Abeta42 oligomers.

Amyloid Beta42 oligomers up‐regulate the excitatory synapses by potentiating presynaptic release while impairing postsynaptic NMDA receptors

Cav1.2 channelopathies causing autism: new hallmarks on Timothy syndrome

Amyloid Beta42 oligomers up‐regulate the excitatory synapses by potentiating presynaptic release while impairing postsynaptic NMDA receptors

Collaborators (10)

Antonia Gurgone

Andrea Marcantoni

Alex Costa

Enis Hidisoglu

Tommaso Pizzorusso

Maurizio Giustetto

Silvia De Rubeis

Luca Palazzolo

Emilio Carbone

Valentina Carabelli

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