Guillermina Lozano

Professor and Chair

The University of Texas MD Anderson Cancer Center

United States

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Guillermina Lozano is a Professor and Chair at The University of Texas MD Anderson Cancer Center in the United States. Her research focuses on the roles of p53 and its mutants in cancer biology, including tumor growth mechanisms and responses to therapies. Recent publications include studies on the inhibition of Mdm2's effects on p53 and the targeted degradation of mutant p53 in lung cancer and triple-negative breast adenocarcinomas.

Articles (17)

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2025

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2021

Collaborators (4)

Richard Gorlick

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Amanda Wasylishen

Assistant Professor

University of Cincinnati

UNITED STATES

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Rasoul Pourebrahim

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Ruli Gao

Assistant Professor

Northwestern University

UNITED STATES

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Guillermina Lozano

Research Interests

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About

Articles (17)

Dependence on Mdm2 for Mdm4 inhibition of p53 activity

Mutant p53 Gain of Function: Why Many See It, Why Some Do Not

Targeted Degradation of Mutant p53 Reverses the Pro-oncogenic Dominant-Negative Effect

Inhibition of ULK1/2 and KRASG12C controls tumor growth in preclinical models of lung cancer

Inhibition of ULK1/2 and KRASG12C controls tumor growth in preclinical models of lung cancer

Mutant p53 protects triple-negative breast adenocarcinomas from ferroptosis in vivo

Unique Transcriptional Profiles Underlie Osteosarcomagenesis Driven by Different p53 Mutants

The histone chaperone function of Daxx is dispensable for embryonic development

Triple-negative breast tumors are dependent on mutant p53 for growth and survival

Tissue specificity and spatio-temporal dynamics of the p53 transcriptional program

p53R245W Mutation Fuels Cancer Initiation and Metastases in NASH-driven Liver Tumorigenesis

Is loss of p53 a driver of ductal carcinoma in situ progression?

p53 Activation Paradoxically Causes Liver Cancer

Context matters – Daxx and Atrx are not robust tumor suppressors in the murine endocrine pancreas

Alterations of the Mdm2 C-Terminus Differentially Impact Its Function In Vivo

Wnt/ß-catenin-mediated p53 suppression is indispensable for osteogenesis of mesenchymal progenitor cells.

Analysis of Population Differences in Digital Conversations About Cancer Clinical Trials: Advanced Data Mining and Extraction Study

Collaborators (4)

Richard Gorlick

Amanda Wasylishen

Rasoul Pourebrahim

Ruli Gao

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