Gunnar Schotta

Professor of Epigenetics

Ludwig-Maximilians-Universität München

Germany

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Gunnar Schotta is a Professor of Epigenetics at Ludwig-Maximilians-Universität München, Germany. His research focuses on the role of epigenetic mechanisms in hematopoiesis, muscle pathology, and cancer metastasis, as well as interactions between DNA methylation and histone modifications. Recent publications include studies on the regulation of gene expression through chromatin dynamics and the impact of somatic mutations on cellular differentiation.

Articles (11)

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2024

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2023

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2021

Collaborators (6)

Andreas Ettinger

Head of Microscopy Core Facility

Helmholtz Zentrum München Deutsches Forschungszentrum für Umwelt und Gesundheit

GERMANY

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Sara Richter

Full Professor

University of Padua

ITALY

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Vladimir Teif

Senior Lecturer

University of Essex

UNITED KINGDOM

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Axel Imhof

Professor for Proteinanalytics

Ludwig-Maximilians-Universität München Medizinische Fakultät

GERMANY

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SARA LAGO

Università degli Studi di Torino

ITALY

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Karsten Rippe

Head of Division

German Cancer Research Center (DKFZ)

GERMANY

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Gunnar Schotta

Research Interests

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About

Articles (11)

Histone methyltransferase SETDB1 safeguards mouse fetal hematopoiesis by suppressing activation of cryptic enhancers

WDR5 is required for DUX4 expression and its pathological effects in FSHD muscular dystrophy

DNA sequence-dependent formation of heterochromatin nanodomains

Dominant role of DNA methylation over H3K9me3 for IAP silencing in endoderm.

HDAC2 Facilitates Pancreatic Cancer Metastasis.

Specific effects of somatic GATA2 zinc finger mutations on erythroid differentiation.

Morc3 silences endogenous retroviruses by enabling Daxx-mediated histone H3.3 incorporation.

Promoter G-quadruplexes and transcription factors cooperate to shape the cell type-specific transcriptome

Epithelial cell plasticity drives endoderm formation during gastrulation.

Suv4-20h2 protects against influenza virus infection by suppression of chromatin loop formation.

Epstein-Barr virus inactivates the transcriptome and disrupts the chromatin architecture of its host cell in the first phase of lytic reactivation

Collaborators (6)

Andreas Ettinger

Sara Richter

Vladimir Teif

Axel Imhof

SARA LAGO

Karsten Rippe

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