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Haiming Wei

University of Science and Technology of China

China

Research Interests

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Professors in Immunology

Articles (20)

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Collaborators (5)

Hong-Di Ma

associate professor

University of Science and Technology of China

CHINA

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Guojun Qian

Professor

Shanghai Jiao Tong University

CHINA

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Guojun Qian

Assoc. Prof

Shanghai Jiao Tong University

CHINA

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Yi Wang

Professor

University of Science and Technology of China

CHINA

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Lin Bai

Principal Investigator

Shanghai Jiao Tong University

CHINA

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Haiming Wei

Research Interests

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Articles (20)

Intrauterine natural killer cell therapy for unexplained recurrent pregnancy failure

Dipeptidylpeptidase 4 inhibition attenuates gestational pathologies via immune homeostasis restoration in the pulmonary-uterine axis

p53: An Immune Ecosystem Manipulator

Correction: CD49a+ NK cells promote esophageal cancer development by inducing MDSCs infiltration via GM-CSF

CD49a+ NK cells promote esophageal cancer development by inducing MDSCs infiltration via GM-CSF

Maternal natural killer cells drive neuroimmune disorders in offspring through aberrant secretion of extracellular granzyme B

Compromised C3b-VSIG4 axis between decidual NK cells and macrophages contributes to recurrent spontaneous abortion

Cytokine Storm Related to CD4+ T Cells in Influenza Virus-Associated Acute Necrotizing Encephalopathy

Tumor-infiltrating platelets promote the growth of lung adenocarcinoma

Sphingomyelin is a prospective metabolic immune checkpoint for natural killer cells

Natural killer cells in tumor immunotherapy

B-cell-derived IL-10 promotes allergic sensitization in asthma regulated by Bcl-3

Blockade of T-cell receptor with Ig and ITIM domains elicits potent antitumor immunity in naturally occurring HBV-related HCC in mice

Erratum: Wang D, Sun Z, Zhu X, et al. GARP-mediated active TGF-β1 induces bone marrow NK cell dysfunction in AML patients with early relapse post–allo-HSCT. Blood. 2022;140(26):2788-2804. (Blood (2022) 140(26) (2788–2804), (S0006497122010643), (10.1182/blood.2022015474))

Interactions between driver genes shape the signaling pathway landscape and direct hepatocellular carcinoma therapy

LCN2 secreted by tissue-infiltrating neutrophils induces the ferroptosis and wasting of adipose and muscle tissues in lung cancer cachexia

Overexpression of lipocalin 2 in PBX1-deficient decidual NK cells promotes inflammation at the maternal-fetal interface

SIRT3-dependent delactylation of cyclin E2 prevents hepatocellular carcinoma growth

The TRIM37 variants in Mulibrey nanism patients paralyze follicular helper T cell differentiation

Tumors evade immune cytotoxicity by altering the surface topology of NK cells

Collaborators (5)

Hong-Di Ma

Guojun Qian

Guojun Qian

Yi Wang

Lin Bai

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