Hanjoong Jo

Wallace H. Coulter Distinguished Chair Professor

georgia institute of technology & emory university

United States

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Hanjoong Jo is the Wallace H. Coulter Distinguished Chair Professor at Emory University and Georgia Institute of Technology in the United States. His research focuses on vascular physiology, atherosclerosis, and cardiovascular diseases, with recent publications addressing topics such as flow-mediated atherosclerosis, transcriptomic heterogeneity in aortic valve cells, and the role of microRNA in endothelial cell function. Through his work, he explores the molecular mechanisms underlying vascular inflammation and remodeling.

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2025

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2021

Collaborators (2)

Eun-Young Erin Ahn

Associate Professor

The University of Alabama

UNITED STATES

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Steve Lim

The University of Alabama

UNITED STATES

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Hanjoong Jo

Research Interests

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Articles (20)

FAK activity exacerbates disturbed flow-mediated atherosclerosis via VEGFR2-CBL-NF-κB signaling

Inhibiting MiR-33a-3p Expression Fails to Enhance ApoAI-Mediated Cholesterol Efflux in Pro-Inflammatory Endothelial Cells

Side- and Disease-Dependent Changes in Human Aortic Valve Cell Population and Transcriptomic Heterogeneity Determined by Single-Cell RNA Sequencing

Saving KLF2/4 from γ-protocadherin to reduce vascular inflammation and atherosclerosis

Publisher Correction: A renaissance of cerebral cavernous malformation proteins in vascular physiology

A renaissance of cerebral cavernous malformation proteins in vascular physiology

A Biomimetic Leaflet Scaffold for Aortic Valve Remodeling.

Editorial: Single-cell OMICs analyses in cardiovascular diseases.

Disturbed flow regulates protein disulfide isomerase A1 expression via microRNA-204.

Hyperoxia impairs induced pluripotent stem cell-derived endothelial cells and drives an atherosclerosis-like transcriptional phenotype.

Nuclear FAK in endothelium: An intrinsic inhibitor of NF-κB activation in atherosclerosis

Flow-induced reprogramming of endothelial cells in atherosclerosis

Spatial control of robust transgene expression in mouse artery endothelium under ultrasound guidance

Hypoxia inducible factor 1α inhibitor PX-478 reduces atherosclerosis in mice

Endothelial Poldip2 regulates sepsis-induced lung injury via Rho pathway activation

Endothelial UCP2 Is a Mechanosensitive Suppressor of Atherosclerosis

Recent Progress in in vitro Models for Atherosclerosis Studies

Stable flow-induced expression of KLK10 inhibits endothelial inflammation and atherosclerosis

Targeting mechanosensitive endothelial TXNDC5 to stabilize eNOS and reduce atherosclerosis in vivo

Conditional Antisense Oligonucleotides Triggered by miRNA

Collaborators (2)

Eun-Young Erin Ahn

Steve Lim

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