Heejung Chun

Assistant Professor

Yonsei University

South Korea

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Heejung Chun is an Assistant Professor at Yonsei University in South Korea. Her research focuses on neurobiology, particularly the role of astrocytes in neurological conditions such as Alzheimer's disease and pain modulation. Recent articles examine the impact of astrocytic functions on cognitive health, glial scar formation, and synaptic plasticity. Dr. Chun’s work contributes to understanding the dynamic interactions within the nervous system and potential therapeutic avenues for neurological disorders.

Articles (10)

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2020

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2020

Collaborators (2)

guido mannaioni

University of Florence

ITALY

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Ja-Hyun Baik

Korea University

SOUTH KOREA

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Heejung Chun

Research Interests

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Articles (10)

Astrocytic autophagy plasticity modulates Aβ clearance and cognitive function in Alzheimer’s disease

KDS2010, a reversible MAO-B inhibitor, extends the lifetime of neural probes by preventing glial scar formation

Role of spinal astrocytes through the perisynaptic astrocytic process in pathological pain

Astrocytes Render Memory Flexible by Releasing D-Serine and Regulating NMDA Receptor Tone in the Hippocampus

Inhibition of monoamine oxidase B prevents reactive astrogliosis and scar formation in stab wound injury model

Longitudinal intravital imaging of cerebral microinfarction reveals a dynamic astrocyte reaction leading to glial scar formation

Erratum: Correction to: Channel-mediated astrocytic glutamate modulates hippocampal synaptic plasticity by activating postsynaptic NMDA receptors (Molecular brain (2015) 8 (7))

Correction: Astrocytic water channel aquaporin-4 modulates brain plasticity in both mice and humans: a potential gliogenetic mechanism underlying language-associated learning (Molecular Psychiatry, (2018), 23, 4, (1021-1030), 10.1038/mp.2017.113)

Dynamic changes in the bridging collaterals of the basal ganglia circuitry control stress-related behaviors in mice

Severe reactive astrocytes precipitate pathological hallmarks of Alzheimer’s disease via H<sub>2</sub>O<sub>2</sub><sup>−</sup> production

Collaborators (2)

guido mannaioni

Ja-Hyun Baik

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