Hisashi Haga

Japan

Research Interests

Explore related searches

Professors in Japan Professors in Tissue Engineering Tissue Engineering professors in Japan

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

Articles (10)

Year:

2024

Year:

2023

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Collaborators (7)

Takuro Ishii

Assistant Professor

Tohoku University

JAPAN

View details

Masayuki Takahashi

Specially Appointed Professor

Hokkaido University

JAPAN

View details

Seiichiro Ishihara

Assistant Professor

Hokkaido University

JAPAN

View details

Kenji Izumi

Professor

Niigata University

JAPAN

View details

Kentaro Suzuki

Wakayama Medical University

JAPAN

View details

Ryoichi Nagatomi

Professor

Tohoku University

JAPAN

View details

Yuji Kumagai

Fred Hutch Cancer Center

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Hisashi Haga

Research Interests

Explore related searches

Contact this professor

Articles (10)

Matrix stiffness regulates the triad communication of adipocytes/macrophages/endothelial cells through CXCL13

Substrate stiffness induces nuclear localization of myosin regulatory light chain to suppress apoptosis

The EGF/EGFR axis and its downstream signaling pathways regulate the motility and proliferation of cultured oral keratinocytes

Stiffness-Modulation of Collagen Gels by Genipin-Crosslinking for Cell Culture.

Improvement of the cell viability of hepatocytes cultured in three-dimensional collagen gels using pump-free perfusion driven by water level difference.

Mammaglobin 1 mediates progression of trastuzumab-resistant breast cancer cells through regulation of cyclins and NF-κB.

The interferon-β/STAT1 axis drives the collective invasion of skin squamous cell carcinoma with sealed intercellular spaces.

Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics.

Matrix Stiffness Contributes to Cancer Progression by Regulating Transcription Factors.

Correction: Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics (Oncogene, (2022), 41, 19, (2764-2777), 10.1038/s41388-022-02288-9)

Collaborators (7)

Takuro Ishii

Masayuki Takahashi

Seiichiro Ishihara

Kenji Izumi

Kentaro Suzuki

Ryoichi Nagatomi

Yuji Kumagai

Make your next move.