Holly Chen

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Assistant Professor

University of Alabama, Tuscaloosa

United States

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About

Dr. Holly Chen is an Assistant Professor at the University of Alabama at Birmingham, USA. Her research focuses on inherited retinal diseases, photoreceptor degeneration, and the development of retinal organoids from pluripotent stem cells. Dr. Chen's recent work includes studies on sex-specific responses to neuroprotective agents and the implications of cell-cell interactions in retinal pathogenesis.

Recent Grants

Grant: Close

Ciliary vesicle loss mediated by CEP290 mutations: implications for ciliogenesis in Leber congenital amaurosis

Open Date: 2025-07-01

Close Date: 2026-06-01

Grant: Close

Restoring extracellular matrix signaling between Müller glia and photoreceptors for therapies of inherited retinal degeneration

Open Date: 2023-07-01

Close Date: 2026-06-01

Articles (10)

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2025

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2025

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2024

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2023

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2022

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2021

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2021

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2020

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2020

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2020

Collaborators (2)

Nicole Morgan

UNITED STATES

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Hyun Beom Song

Associate Professor

Seoul National University

SOUTH KOREA

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Holly Chen

Research Interests

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About

Recent Grants

Articles (10)

Sex-specific attenuation of photoreceptor degeneration by reserpine in a rhodopsin P23H rat model of autosomal dominant retinitis pigmentosa

Sex-specific attenuation of photoreceptor degeneration by reserpine in a rhodopsin P23H rat model of autosomal dominant retinitis pigmentosa

Cell-cell interaction in the pathogenesis of inherited retinal diseases.

Reserpine maintains photoreceptor survival in retinal ciliopathy by resolving proteostasis imbalance and ciliogenesis defects

Nicotinamide Promotes Formation of Retinal Organoids From Human Pluripotent Stem Cells <i>via</i> Enhanced Neural Cell Fate Commitment.

Erratum: Accelerated and Improved Differentiation of Retinal Organoids from Pluripotent Stem Cells in Rotating-Wall Vessel Bioreactors (Stem Cell Reports (2018) 10(1) (300–313), (S2213671117304873), (10.1016/j.stemcr.2017.11.001))

Genetics and therapy for pediatric eye diseases.

Primary cilia biogenesis and associated retinal ciliopathies.

Accelerated Development of Rod Photoreceptors in Retinal Organoids Derived from Human Pluripotent Stem Cells by Supplementation with 9-<i>cis</i> Retinal.

HIPRO: A High-Efficiency, Hypoxia-Induced Protocol for Generation of Photoreceptors in Retinal Organoids from Mouse Pluripotent Stem Cells.

Collaborators (2)

Nicole Morgan

Hyun Beom Song

Make your next move.

Holly Chen

Research Interests

Explore related searches

Contact this professor

About

Recent Grants

Articles (10)

Sex-specific attenuation of photoreceptor degeneration by reserpine in a rhodopsin P23H rat model of autosomal dominant retinitis pigmentosa

Sex-specific attenuation of photoreceptor degeneration by reserpine in a rhodopsin P23H rat model of autosomal dominant retinitis pigmentosa

Cell-cell interaction in the pathogenesis of inherited retinal diseases.

Reserpine maintains photoreceptor survival in retinal ciliopathy by resolving proteostasis imbalance and ciliogenesis defects

Nicotinamide Promotes Formation of Retinal Organoids From Human Pluripotent Stem Cells <i>via</i> Enhanced Neural Cell Fate Commitment.

Erratum: Accelerated and Improved Differentiation of Retinal Organoids from Pluripotent Stem Cells in Rotating-Wall Vessel Bioreactors (Stem Cell Reports (2018) 10(1) (300–313), (S2213671117304873), (10.1016/j.stemcr.2017.11.001))

Genetics and therapy for pediatric eye diseases.

Primary cilia biogenesis and associated retinal ciliopathies.

Accelerated Development of Rod Photoreceptors in Retinal Organoids Derived from Human Pluripotent Stem Cells by Supplementation with 9-&lt;i&gt;cis&lt;/i&gt; Retinal.

HIPRO: A High-Efficiency, Hypoxia-Induced Protocol for Generation of Photoreceptors in Retinal Organoids from Mouse Pluripotent Stem Cells.

Collaborators (2)

Nicole Morgan

Hyun Beom Song

Make your next move.

Accelerated Development of Rod Photoreceptors in Retinal Organoids Derived from Human Pluripotent Stem Cells by Supplementation with 9-<i>cis</i> Retinal.