Ibra Fancher

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Assistant Professor

University of Delaware

United States

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About

Ibra Fancher is an Assistant Professor at the University of Delaware in the United States. His research focuses on the mechanisms of endothelial dysfunction related to obesity, hypertension, and metabolic conditions. Recent articles authored by Dr. Fancher explore topics such as the impact of adipose tissue on endothelial function and cardiovascular health, as well as T-cell metabolism in midlife adults.

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Obesity and Endothelial Dsyfunction: Kir channels in the progression of cardiovascular disease

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Articles (15)

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2025

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2021

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2020

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2020

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2020

Collaborators (15)

Orlando M. Gutierrez

The University of Alabama

UNITED STATES

Ibra Fancher

Research Interests

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About

Recent Grants

Articles (15)

Inhibition of lipolysis in visceral adipose tissue from obese mice and humans prevents impairment of endothelial Kir2.1 channels

Elevated LDL-C induces T-cell metabolic dysfunction and increases inflammation and oxidative stress in midlife adults

CD8+ T-cell metabolism is related to cerebrovascular reactivity in middle-aged adults

Visceral adipose of obese mice inhibits endothelial inwardly rectifying K+ channels in a CD36-dependent fashion

A long-term high-fat diet induces differential gene expression changes in spatially distinct adipose tissue of male mice

Impairment of microvascular endothelial Kir2.1 channels contributes to endothelial dysfunction in human hypertension

The damaging duo: Obesity and excess dietary salt contribute to hypertension and cardiovascular disease

Membrane Cholesterol Interactions with Proteins in Hypercholesterolemia-Induced Endothelial Dysfunction

New developments in translational microcirculatory research

Cholesterol-Induced Suppression of Endothelial Kir Channels Is a Driver of Impairment of Arteriolar Flow-Induced Vasodilation in Humans

Differential effects of obesity on visceral versus subcutaneous adipose arteries: role of shear-activated Kir2.1 and alterations to the glycocalyx

The physiological benefits of sitting less and moving more: Opportunities for future research.

Impairment of Flow-Sensitive Inwardly Rectifying K<sup>+</sup> Channels via Disruption of Glycocalyx Mediates Obesity-Induced Endothelial Dysfunction.

Endothelial inwardly-rectifying K<sup>+</sup> channels as a key component of shear stress-induced mechanotransduction.

LDL induces cholesterol loading and inhibits endothelial proliferation and angiogenesis in Matrigels: correlation with impaired angiogenesis during wound healing.

Collaborators (15)

Orlando M. Gutierrez

Kishore Wary

Austin Robinson

Andreas Beyer

James Lee

Christopher Martens

Joseph Watso

Irena Levitan

David Edwards

Sang Joon Ahn

Julie K Freed

Luisa Ann DiPietro

Amanda LeBlanc

Teresa Chen

Chueh-Lung Hwang

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