Ignacio García-Tuñón Llanio

Profesor Ayudante Doctor

Universidad de Alcalá

Spain

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About

Ignacio García-Tuñón Llanio is an Assistant Professor at Universidad de Alcalá in Spain. His research areas encompass cancer biology, genetic variants related to hematological disorders, and the application of CRISPR/Cas9 technology in developing therapeutic strategies. Recent articles authored by him explore topics such as the impact of genetic modifications on tumorigenesis and the relationship between chronic venous disease and inflammation during pregnancy.

Articles (14)

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2025

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2023

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2023

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2023

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2021

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2020

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2020

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2020

Collaborators (23)

Jesús María Hernández-Rivas

Universidad de Salamanca

SPAIN

Ignacio García-Tuñón Llanio

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Articles (14)

Examining the Effects of the RUNX1 p.Leu43Ser Variant on FPD/AML Phenotypes Using a CRISPR/Cas9-Generated Knock-In Murine Model

Histone Acetyl Transferase 1 Is Overexpressed in Poor Prognosis, High-grade Meningeal and Glial Brain Cancers: Immunohistochemical and Aptahistochemical Study

Chronic Venous Disease during Pregnancy Is Related to Inflammation of the Umbilical Cord: Role of Allograft Inflammatory Factor 1 (AIF-1) and Interleukins 10 (IL-10), IL-12 and IL-18

Understanding HAT1: A Comprehensive Review of Noncanonical Roles and Connection with Disease

Novel variants in GALE cause syndromic macrothrombocytopenia by disrupting glycosylation and thrombopoiesis

METTL1 promotes tumorigenesis through tRNA-derived fragment biogenesis in prostate cancer.

A Potential Effect of Circadian Rhythm in the Delivery/Therapeutic Performance of Paclitaxel–Dendrimer Nanosystems

CRISPR/Cas9-Directed Gene Trap Constitutes a Selection System for Corrected BCR/ABL Leukemic Cells in CML

A novel nonsense variant in TPM4 caused dominant macrothrombocytopenia, mild bleeding tendency and disrupted cytoskeleton remodeling.

Granuloma Formation in a <i>Cyba</i>-Deficient Model of Chronic Granulomatous Disease Is Associated with Myeloid Hyperplasia and the Exhaustion of B-Cell Lineage

CRISPR-Cas9 Technology as a Tool to Target Gene Drivers in Cancer: Proof of Concept and New Opportunities to Treat Chronic Myeloid Leukemia

Loss of HDAC11 accelerates skeletal muscle regeneration.

CRISPR/Cas9-generated models uncover therapeutic vulnerabilities of del(11q) CLL cells to dual BCR and PARP inhibition

ETV6/RUNX1 Fusion Gene Abrogation Decreases the Oncogenicity of Tumour Cells in a Preclinical Model of Acute Lymphoblastic Leukaemia.

Collaborators (23)

Jesús María Hernández-Rivas

Diana Costa

Miguel A. Ortega

Ana Herrero

Angelika Merkel

Telma Quintela

Adrián Montaño

Manuel Sanchez-Martin

Vittorio Abbonante

Angel Hernandez-Hernandez

Melchor Álvarez-Mon

Mar Royuela

Juan A. De León-Luis

José Rivera

Óscar Fraile Martínez

Toledo Lobo M. Val

José María Bastida

Miguel A. Saez

M ROCIO BENITO SANCHEZ

Michaela Essl-Gruber

Natalio Garcia Honduvilla

Jesús Sánchez-Yagüe

Javier De Las Rivas

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