Isabella Rauch

Assistant Professor

Oregon Health & Science University

United States

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About

Isabella Rauch is an Assistant Professor at Oregon Health and Science University in the United States. Her research focuses on the immunological interactions between host epithelial cells and pathogens, particularly concerning inflammasome activation and innate immune responses. Recent publications highlight her work on the protective effects of microbial extracellular vesicles and the mechanisms of epithelial cell immunity against bacterial infections.

Articles (16)

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2025

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2020

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2020

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2020

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2020

Collaborators (6)

Oyebola Oyesola

National Institutes of Health

UNITED STATES

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Russell E Vance

University of California

UNITED STATES

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Justin Merritt

Professor

Oregon Health & Science University

UNITED STATES

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Timothy Nice

Associate Professor

Oregon Health & Science University

UNITED STATES

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Sunny Shin

University of Pennsylvania

UNITED STATES

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Leigh Knodler

Associate Professor

University of Vermont

UNITED STATES

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Isabella Rauch

Research Interests

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Articles (16)

Immunostimulatory effects of Streptococcus sanguinis extracellular membrane vesicles protect oral gingival epithelial cells from periodontal pathobiont damage

Enteric tuft cell inflammasome activation drives NKp46+ILC3 IL22 via PGD2 and inhibits Salmonella.

Why put yourself on a pedestal? The pathogenic role of the A/E pedestal

Interferon regulatory factor 6 (IRF6) determines intestinal epithelial cell development and immunity

Inducing Pyroptosis with FlaTox, RodTox, or NeedleTox.

Epithelial Pyroptosis in Host Defense

Human NAIP/NLRC4 and NLRP3 inflammasomes detect Salmonella type III secretion system activities to restrict intracellular bacterial replication.

Salmonella enterica Serovar Typhimurium Induces NAIP/NLRC4- and NLRP3/ASC-Independent, Caspase-4-Dependent Inflammasome Activation in Human Intestinal Epithelial Cells

Inflammasome activation leads to cDC1-independent cross-priming of CD8 T cells by epithelial cell-derived antigen.

Innate immune sensing by epithelial barriers.

PGD2 and CRTH2 counteract Type 2 cytokine-elicited intestinal epithelial responses during helminth infection.

Lytic Cell Death in Specific Microglial Subsets Is Required for Preventing Atypical Behavior in Mice.

NAIP-NLRC4-deficient mice are susceptible to shigellosis.

NLRC4 inflammasome activation is NLRP3- and phosphorylation-independent during infection and does not protect from melanoma

The NAIP/NLRC4 inflammasome in infection and pathology.

Intestinal restriction of Salmonella Typhimurium requires caspase-1 and caspase-11 epithelial intrinsic inflammasomes

Collaborators (6)

Oyebola Oyesola

Russell E Vance

Justin Merritt

Timothy Nice

Sunny Shin

Leigh Knodler

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