Ivan Luptak

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Boston University

United States

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Elevated intracellular sodium worsens mitochondrial ROS and ATP production in obesity induced diastolic dysfunction.

Open Date: 2015-07-01

Close Date: 2020-06-01

Articles (9)

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2023

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2022

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2022

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2021

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2020

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2020

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2020

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2020

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2020

Collaborators (1)

Fedor Simko

SLOVAKIA

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Ivan Luptak

Research Interests

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Recent Grants

Articles (9)

Multi-Omics Profiling of Hypertrophic Cardiomyopathy Reveals Altered Mechanisms in Mitochondrial Dynamics and Excitation–Contraction Coupling

Ivabradine in the Management of COVID-19-related Cardiovascular Complications: A Perspective

Novel Small-Molecule Troponin Activator Increases Cardiac Contractile Function Without Negative Impact on Energetics

Effects of Sodium-Glucose Linked Transporter 2 Inhibition With Ertugliflozin on Mitochondrial Function, Energetics, and Metabolic Gene Expression in the Presence and Absence of Diabetes Mellitus in Mice.

Redox-Resistant SERCA [Sarco(endo)plasmic Reticulum Calcium ATPase] Attenuates Oxidant-Stimulated Mitochondrial Calcium and Apoptosis in Cardiac Myocytes and Pressure Overload-Induced Myocardial Failure in Mice.

Role of Glutaredoxin-1 and Glutathionylation in Cardiovascular Diseases.

Differential Effects of Sacubitril/Valsartan on Diastolic Function in Mice With Obesity-Related Metabolic Heart Disease.

A novel tracer for in vivo optical imaging of fatty acid metabolism in the heart and brown adipose tissue.

Increasing mitochondrial ATP synthesis with butyrate normalizes ADP and contractile function in metabolic heart disease

Collaborators (1)

Fedor Simko

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