Jae-Young Je

Professor

Pusan National University

South Korea

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Professor Jae-Young Je is a faculty member at Pukyong National University in South Korea. His research focuses on cardiovascular disease, obesity, and the therapeutic potential of bioactive peptides derived from marine resources. Recent publications highlight his work on the effects of various peptides on endothelial dysfunction and adipocyte browning, contributing to advancements in understanding metabolic disorders.

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2026

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2020

Collaborators (4)

Chang-Hyung Choi

Yeungnam University

SOUTH KOREA

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Young-Mog Kim

Pusan National University

SOUTH KOREA

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Hyun Wook Kang

SOUTH KOREA

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Won-Kyo Jung

Prof.

Pusan National University

SOUTH KOREA

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Jae-Young Je

Research Interests

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About

Articles (25)

Ark shell-derived peptides AWLNH (P3) and PHDL (P4) as novel inhibitors of endothelial dysfunction in Cardiovascular disease models

Lotus seed (Nelumbo nucifera) protein-derived calcium-binding peptides: Isolation, characterization, and osteogenic effect

Glutathione as a Therapeutic Agent for OxLDL‐Induced Endothelial Dysfunction and Atherosclerosis Prevention

Salicornia europaea L. Ethyl Acetate Fraction Induces Adipocyte Browning and Exhibits Anti‐Obesity Effect in 3T3‐L1 Preadipocytes

Fucosterol Promotes Browning in Mouse 3T3‐L1 Adipocytes Through HO‐1/Nrf2 and AMPK Pathways

Multicompartmental Hydrogel Microspheres with a Concentric Thin Oil Layer: Protecting and Targeting Therapeutic Agents for Inflammatory Bowel Disease

Ark Shell-Derived Peptides AWLNH (P3) and PHDL (P4) Mitigate Foam Cell Formation by Modulating Cholesterol Metabolism and HO-1/Nrf2-Mediated Oxidative Stress in Atherosclerosis

Oral administration of PIISVYWK and FSVVPSPK peptides attenuates obesity, oxidative stress, and inflammation in high fat diet-induced obese mice

Neptunea cumingii Extract Inhibits Adipogenesis and Stimulates Browning of Adipocytes in 3T3‐L1 Preadipocytes

Mussels as sustainable marine resources for bioactive peptides for health and the food industry

Brown Algae Ecklonia cava Extract Modulates Adipogenesis and Browning in 3T3-L1 Preadipocytes through HO-1/Nrf2 Signaling

Two peptides LLRLTDL and GYALPCDCL inhibit foam cell formation through activating PPAR‐γ/LXR‐α signaling pathway in oxLDL‐treated RAW264.7 macrophages

Brown Algae <i>Dictyopteris divaricata</i> Attenuates Adipogenesis by Modulating Adipocyte Differentiation and Promoting Lipolysis through Heme Oxygenase-1 Activation in 3T3-L1 Cells

Multifunctional hydrogel dressing based on fish gelatin/oxidized hyaluronate for promoting diabetic wound healing

Bioactive peptides from food proteins as potential anti-obesity agents: Mechanisms of action and future perspectives

Photothermally controlled drug release of naproxen-incorporated mungbean starch/PVA biomaterials adding melanin nanoparticles

OxLDL-Induced Foam Cell Formation Inhibitory Activity of Pepsin Hydrolysate of Ark Shell (Scapharca subcrenata (Lischke, 1869)) in RAW264.7 Macrophages

Protective effect of multifunctional peptides PIISVYWK and FSVVPSPK on oxidative stress-mediated HUVEC injury through antioxidant and anti-apoptotic action

Blue mussel (Mytilus edulis) hydrolysates attenuate oxidized-low density lipoproteins (ox-LDL)-induced foam cell formation, inflammation, and oxidative stress in RAW264.7 macrophages

Anti‐inflammatory action of ark shell ( Scapharca subcrenata ) protein hydrolysate in LPS‐stimulated RAW264.7 murine macrophages

Marine Biological Macromolecules and Chemically Modified Macromolecules; Potential Anticoagulants

Heme oxygenase‐1 induction by gallic acid‐ g ‐chitosan is an important event in modulating adipocyte differentiation

Cytoprotective Peptides from Blue Mussel Protein Hydrolysates: Identification and Mechanism Investigation in Human Umbilical Vein Endothelial Cells Injury

Cytoprotective Role of Edible Seahorse (Hippocampus abdominalis)-Derived Peptides in H2O2-Induced Oxidative Stress in Human Umbilical Vein Endothelial Cells

Ark shell protein‐derived bioactive peptides promote osteoblastic differentiation through upregulation of the canonical Wnt/β‐catenin signaling in human bone marrow‐derived mesenchymal stem cells

Collaborators (4)

Chang-Hyung Choi

Young-Mog Kim

Hyun Wook Kang

Won-Kyo Jung

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