James West

Professor

Vanderbilt University Medical Center

United States

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James West is a Professor at Vanderbilt University Medical Center in the United States. His research focuses on pulmonary arterial hypertension, heart failure, and the molecular mechanisms of cardiac and vascular remodeling. Recent articles highlight his work on transcriptional signatures in heart failure and the role of various cellular mechanisms in pulmonary vascular diseases.

Articles (23)

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2020

Collaborators (4)

Anandharajan Rathinasabapathy

Research Assistant Professor

Vanderbilt University Medical Center

UNITED STATES

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Stephen Halliday

Assistant Professor

University of Wisconsin-Madison

UNITED STATES

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Sergey Gutor

Research Assistant Professor

Vanderbilt University Medical Center

UNITED STATES

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Jonathan Kropski

Associate Professor

Vanderbilt University Medical Center

UNITED STATES

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James West

Research Interests

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Articles (23)

Mas1 Receptor Activation is Necessary and Sufficient to Transduce ACE2 Effect in PAH, But Ang(1‐7) Alone is Insufficient

Transcriptional Signatures of the Right Ventricle in End‐Stage Heart Failure

RNA‐Seq and ChIP‐Seq Identification of Unique and Overlapping Target Genes and Pathways Regulated by TBX4 in Human Pulmonary Fibroblasts and Pericytes

Repetitive sulfur dioxide exposure in mice models post-deployment respiratory syndrome

A multimodal approach identifies lactate as a central feature of right ventricular failure that is detectable in human plasma

Carnitine consumption and effect of oral supplementation in human pulmonary arterial hypertension: A pilot study

Molecular Function and Contribution of <i>TBX4</i> in Development and Disease

Myeloid Cell Derived IL1b Contributes to Pulmonary Vascular Remodeling in Heart Failure with Preserved Ejection Fraction

Myeloid Cell Derived IL1β Contributes to Pulmonary Hypertension in HFpEF

Repetitive Sulfur Dioxide Exposure in Mice Models Post-Deployment Respiratory Syndrome

A potential adverse role for leptin and cardiac leptin receptor in the right ventricle in pulmonary arterial hypertension: effect of metformin is BMPR2 mutation-specific

Myeloid Cell Derived IL1b Contributes to Pulmonary Vascular Remodeling in Heart Failure With Preserved Ejection Fraction

Blockade Or Deletion Of The Thromboxane/Prostanoid Receptor Reduces Right Ventricular Stiffness To Maintain Function In Right Ventricular Pressure Overload

Loss of talin in cardiac fibroblasts results in augmented ventricular cardiomyocyte hypertrophy in response to pressure overload

Systems-level regulation of microRNA networks by miR-130/301 promotes pulmonary hypertension (vol 124, pg 3514, 2014)

Thromboxane-Prostanoid Receptor Signaling Drives Persistent Fibroblast Activation in Pulmonary Fibrosis

l-Carnitine therapy improves right heart dysfunction through Cpt1-dependent fatty acid oxidation

TBX4 Transcription Factor Is a Positive Feedback Regulator of Itself and Phospho-SMAD1/5.

BMP9 in ARDS - Decades of BMP Studies in Vascular Biology Paying Off?

Mechanistic Phase II Clinical Trial of Metformin in Pulmonary Arterial Hypertension.

Expression of a Human Caveolin-1 Mutation in Mice Drives Inflammatory and Metabolic Defect-Associated Pulmonary Arterial Hypertension.

Resident mesenchymal vascular progenitors modulate adaptive angiogenesis and pulmonary remodeling via regulation of canonical Wnt signaling.

A multifaceted investigation into molecular associations of chronic thromboembolic pulmonary hypertension pathogenesis.

Collaborators (4)

Anandharajan Rathinasabapathy

Stephen Halliday

Sergey Gutor

Jonathan Kropski

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