Jan-Philipp Mallm

Head

Deutsches Krebsforschungszentrum

Germany

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About

Jan-Philipp Mallm is the Head at the Deutsches Krebsforschungszentrum in Germany. His research areas focus on medulloblastoma progression, the genomic consequences of chromothripsis, and single-cell RNA sequencing techniques. Recent articles highlight his work on oncogene aberrations, glioblastoma cell-state plasticity, and the epigenetic signals in cellular responses. He is also investigating the microenvironmental impacts on multiple myeloma and the heterogeneity of DNA methylation landscapes.

Articles (14)

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2025

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2021

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2021

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2021

Collaborators (7)

Nick Kepper

Heidelberg University

GERMANY

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Kristina Schoonjans

Associate Professor

École Polytechnique Fédérale de Lausanne

SWITZERLAND

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Medhanie A. Mulaw

Professor (Molecular Oncology and Functional Genomics)

Universität Ulm

GERMANY

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Karsten Rippe

Head of Division

German Cancer Research Center (DKFZ)

GERMANY

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Fabian Erdel

Université de Toulouse

FRANCE

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Felix Meissner

Full Professor

University of Bonn

GERMANY

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Ann Daly

Professor

Newcastle University

UNITED KINGDOM

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Jan-Philipp Mallm

Research Interests

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Articles (14)

Oncogene aberrations drive medulloblastoma progression, not initiation

Author Correction: Multi-omic and single-cell profiling of chromothriptic medulloblastoma reveals genomic and transcriptomic consequences of genome instability

SOX10 mediates glioblastoma cell-state plasticity

Spatio-temporal transcriptomics of chromothriptic SHH-medulloblastoma identifies multiple genetic clones that resist treatment and drive relapse

Multi-omic and single-cell profiling of chromothriptic medulloblastoma reveals genomic and transcriptomic consequences of genome instability

Epigenetic signals that direct cell type–specific interferon beta response in mouse cells

Approaches for single-cell RNA sequencing across tissues and cell types

Reduced adhesion of aged intestinal stem cells contributes to an accelerated clonal drift.

Downregulation of TGR5 (GPBAR1) in biliary epithelial cells contributes to the pathogenesis of sclerosing cholangitis.

NASH limits anti-tumour surveillance in immunotherapy-treated HCC.

Subclone-specific microenvironmental impact and drug response in refractory multiple myeloma revealed by single-cell transcriptomics.

The DNA methylation landscape of multiple myeloma shows extensive inter- and intrapatient heterogeneity that fuels transcriptomic variability.

Author Correction: Auto-aggressive CXCR6<sup>+</sup> CD8 T cells cause liver immune pathology in NASH.

Auto-aggressive CXCR6<sup>+</sup> CD8 T cells cause liver immune pathology in NASH.

Collaborators (7)

Nick Kepper

Kristina Schoonjans

Medhanie A. Mulaw

Karsten Rippe

Fabian Erdel

Felix Meissner

Ann Daly

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