Jaroslav Truksa

Lab Head, Principal Investigator

Czech Academy of Sciences

Czech Republic

Research Interests

Explore related searches

Professors in Cancer Biology

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Jaroslav Truksa is a Lab Head and Principal Investigator at the Czech Academy of Sciences. His research primarily focuses on mitochondrial iron metabolism, antiparasitic strategies, and anticancer therapies, as evidenced by his recent publications. His work includes studies on the efficacy of third-generation taxanes in various cancer models and the application of CRISPR/Cas9 technology in genetic research.

Articles (8)

Year:

2024

Year:

2024

Year:

2023

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2020

Collaborators (5)

Dominik Arbon

Head of Unit

CZECH REPUBLIC

View details

Alzbeta Spalenkova

Charles University, Faculty of Medicine in Pilsen

CZECH REPUBLIC

View details

Ludmila Krizova

Charles University

CZECH REPUBLIC

View details

Martin Vokurka

Dean

Charles University

CZECH REPUBLIC

View details

Petr Prikryl

Charles University, First Faculty of Medicine

CZECH REPUBLIC

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Jaroslav Truksa

Research Interests

Explore related searches

Contact this professor

About

Articles (8)

Third-generation taxanes SB-T-121605 and SB-T-121606 are effective in pancreatic ductal adenocarcinoma

Chelation of Mitochondrial Iron as an Antiparasitic Strategy

Mitochondrially targeted tamoxifen as anticancer therapy: case series of patients with renal cell carcinoma treated in a phase I/Ib clinical trial

Anticancer regimens containing third generation taxanes SB-T-121605 and SB-T-121606 are highly effective in resistant ovarian carcinoma model

Generation and characterization of human U-2 OS cell lines with the CRISPR/Cas9-edited protoporphyrinogen oxidase IX gene

Matriptase-2 and Hemojuvelin in Hepcidin Regulation: In Vivo Immunoblot Studies in <i>Mask</i> Mice

Targeting mitochondrial iron metabolism suppresses tumor growth and metastasis by inducing mitochondrial dysfunction and mitophagy.

Complex Mitochondrial Dysfunction Induced by TPP+-Gentisic Acid and Mitochondrial Translation Inhibition by Doxycycline Evokes Synergistic Lethality in Breast Cancer Cells.

Collaborators (5)

Dominik Arbon

Alzbeta Spalenkova

Ludmila Krizova

Martin Vokurka

Petr Prikryl

Make your next move.