Jean-Sebastien Silvestre

Academic Professor

Institut national de la santé et de la recherche médicale

France

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About

Jean-Sebastien Silvestre is an Academic Professor at the Institut national de la santé et de la recherche médicale in France. His research focuses on cardiovascular biology, specifically exploring the role of regulatory T cells and autophagy in cardiac health. His recent publications include studies on stem cell therapies for myocardial infarction and the therapeutic potential of extracellular vesicles derived from cardiac progenitor cells.

Articles (16)

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2025

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (18)

Ludivine Laurans

INSERM

FRANCE

Jean-Sebastien Silvestre

Research Interests

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Articles (16)

Regulatory T Cells Boost Efficacy of Post-Infarction Pluripotent Stem Cell-Derived Cardiovascular Progenitor Cell Transplants

Prostaglandins bet on regulatory T cells to promote therapeutic revascularization

Autophagy protein 5 controls flow-dependent endothelial functions

Genetic inhibition of CARD9 accelerates the development of atherosclerosis in mice through CD36 dependent-defective autophagy

Correction: Autophagy protein 5 controls flow-dependent endothelial functions.

Therapeutic potential of extracellular vesicles derived from cardiac progenitor cells in rodent models of chemotherapy-induced cardiomyopathy.

Biomaterial-embedded extracellular vesicles improve recovery of the dysfunctional myocardium

Dynamics of monocyte-derived macrophage diversity in experimental myocardial infarction.

Rôle délétère des lymphocytes B de la zone marginale de la rate dans le remodelage ventriculaire après un infarctus du myocarde

Identification of Adipose Tissue as a Reservoir of Macrophages after Acute Myocardial Infarction

Emerging Roles of the Atypical Chemokine Receptor 3 (ACKR3) in Cardiovascular Diseases.

Splenic Marginal Zone B Lymphocytes Regulate Cardiac Remodeling After Acute Myocardial Infarction in Mice.

Extracellular vesicles fail to trigger the generation of new cardiomyocytes in chronically infarcted hearts.

Innate Lymphoid Cells Promote Recovery of Ventricular Function After Myocardial Infarction.

Cytotoxic CD8<sup>+</sup> T cells promote granzyme B-dependent adverse post-ischemic cardiac remodeling.

Modeling Acute Pericarditis

Collaborators (18)

Ludivine Laurans

Harry Sokol

Corinne Tanchot

Ziad Mallat

Paul Alayrac

Simon Bond

Marcella Ma

Etienne Roux

Pascal Reynier

Olivia Lenoir

Joseph Cheriyan

Tian Zhao

Antoine-Emmanuel Saliba

Alma Zernecke

Angelique Levoye

Pierre-Louis Tharaux

Audrey Cras

Joern Dengjel

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