Jeffery Tessem

Associate Professor

Brigham Young University

United States

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Jeffery Tessem is an Associate Professor at Brigham Young University in the United States. His research primarily focuses on pancreatic beta cell function, insulin secretion, and metabolic health, as evidenced by recent articles exploring β-cell adaptation and the impact of dietary compounds. He is dedicated to advancing the understanding of diabetes pathogenesis and the role of microbiota-derived metabolites in metabolic regulation.

Articles (14)

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (4)

Susan M. Smith

UNITED STATES

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Yumi Imai

Iowa State University

UNITED STATES

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Andrew Neilson

Associate Professor

North Carolina State University

UNITED STATES

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Scott Soleimanpour

University of Michigan

UNITED STATES

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Jeffery Tessem

Research Interests

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Articles (14)

Pancreatic islet adaptation in pregnancy and postpartum

CEBPA Overexpression Enhances β-Cell Proliferation and Survival

Loss of glucose-stimulated β-cell Nr4a1 expression impairs insulin secretion and glucose homeostasis

Editorial: Study of pancreatic islets based on human models to understand pathogenesis of diabetes

Bioavailable Microbial Metabolites of Flavanols Demonstrate Highly Individualized Bioactivity on In Vitro β-Cell Functions Critical for Metabolic Health

Cocoa extract exerts sex-specific anti-diabetic effects in an aggressive type-2 diabetes model: A pilot study

Ca2+ Sensors Assemble: Function of the MCU Complex in the Pancreatic Beta Cell

Potential of Phenolic Compounds and Their Gut Microbiota-Derived Metabolites to Reduce TMA Formation: Application of an In Vitro Fermentation High-Throughput Screening Model

Gut Metabolite Trimethylamine N-Oxide Protects INS-1 β-Cell and Rat Islet Function under Diabetic Glucolipotoxic Conditions

Decreased proliferation of aged rat beta cells corresponds with enhanced expression of the cell cycle inhibitor p27KIP1

The Accumulation and Molecular Effects of Trimethylamine N-Oxide on Metabolic Tissues: It’s Not All Bad

Diet-induced obesity in genetically diverse collaborative cross mouse founder strains reveals diverse phenotype response and amelioration by quercetin treatment in 129S1/SvImJ, PWK/EiJ, CAST/PhJ, and WSB/EiJ mice

Good Cop, Bad Cop: The Opposing Effects of Macrophage Activation State on Maintaining or Damaging Functional β-Cell Mass

Lack of skeletal muscle liver kinase B1 alters gene expression, mitochondrial content, inflammation and oxidative stress without affecting high-fat diet-induced obesity or insulin resistance.

Collaborators (4)

Susan M. Smith

Yumi Imai

Andrew Neilson

Scott Soleimanpour

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