Jeffrey Gagan

Assistant Professor

University of Texas Southwestern Medical School

United States

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Dr. Jeffrey Gagan is an Assistant Professor at the University of Texas Southwestern Medical School in the United States. His research areas include molecular pathology, renal tumors, and comprehensive genomic characterization of various cancers, as highlighted in his recent publications. Dr. Gagan's work focuses on the application of large language models in pathology, the molecular testing challenges in clinical practices, and the identification of novel fusion events in tumors.

Articles (10)

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2024

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2023

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2023

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2023

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2023

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2023

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2023

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2023

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2023

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2023

Collaborators (3)

Pawel Mroz

University of Minnesota

UNITED STATES

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Wendong Yu

Associate Professor

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Audrey Jajosky

University of Michigan

UNITED STATES

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Jeffrey Gagan

Research Interests

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Articles (10)

The Potential Utility of Large Language Models in Molecular Pathology

Adamantinoma-like Ewing Sarcoma (ALES) May Harbor FUS Rearrangements: A Potential Diagnostic Pitfall.

Current clinical practices and challenges in molecular testing

Multicystic Clear Cell Renal Tumors With Low-grade Nuclear Features: Time to Include TFE3 Translocation-associated Carcinomas.

Comprehensive Next-Generation Sequencing Testing in a Patient with TEMPI Syndrome.

Comprehensive Molecular Characterization of Polymorphous Adenocarcinoma, Cribriform Subtype: Identifying Novel Fusions and Fusion Partners.

"Oncocytoid Renal Cell Carcinomas After Neuroblastoma" Represent TSC -mutated Eosinophilic Solid and Cystic Renal Cell Carcinomas : Association With Prior Childhood Malignancy and Multifocality With Therapeutic Implications.

Palisading Adenocarcinoma: A Morphologically Unique Salivary Gland Tumor With a Neuroendocrine-like Appearance and a Predilection for the Sublingual Glands of Women.

Refining the serum miR-371a-3p test for viable germ cell tumor detection.

Presence but not number of secondary type mutations influences outcome in de novo AML without MDS-associated or recurring cytogenetic abnormalities.

Collaborators (3)

Pawel Mroz

Wendong Yu

Audrey Jajosky

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