Jens Schuster

Assistant Professor

Uppsala University

Sweden

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Jens Schuster is an Assistant Professor at Uppsala Universitet in Sweden. His research focuses on neural network abnormalities and epigenetic factors in developmental disorders, particularly related to Dravet syndrome and other genetic conditions. He has contributed to the understanding of GABAergic development and the functional implications of induced pluripotent stem cells in various syndromic disorders.

Articles (12)

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2025

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2024

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2024

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2022

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2021

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2021

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2021

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2020

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2020

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2020

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2020

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2020

Collaborators (7)

Joey Lau

Senior Lecturer

Uppsala University

SWEDEN

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Marc Friedlander

SciLifeLab Group Leader, head of Quantitative Biology Group

SWEDEN

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Andrey Vinogradov

Tampere University

FINLAND

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xingqi chen

Associate professor of molecular medicine, group leader

Uppsala University

SWEDEN

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Bryndis Birnir

Professor of Physiology (Chair)

Uppsala University

SWEDEN

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Tanja Hyvärinen

University of Tampere

FINLAND

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Niklas Dahl

Professor and Senior consultant in Clinical Genetics

Uppsala University

SWEDEN

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Jens Schuster

Research Interests

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About

Articles (12)

Abnormalities in the functional activity of neural networks in a human iPSC model of Dravet syndrome

Epigenetic insights into GABAergic development in Dravet Syndrome iPSC and therapeutic implications

Epigenetic insights into GABAergic development in Dravet Syndrome iPSC and therapeutic implications

ZEB2 haploinsufficient Mowat-Wilson syndrome induced pluripotent stem cells show disrupted GABAergic transcriptional regulation and function.

Generation of human induced pluripotent stem cell (iPSC) lines (UUMCBi001-A, UUMCBi002-A) from two healthy donors

Syndromic RNA polymerase II insufficiency: Generation of a human induced pluripotent stem cell line (UUIGPi002A-5) with a heterozygous disruption of POLR2A

A combined approach for single-cell mRNA and intracellular protein expression analysis

Generation of two human iPSC lines (UUIGPi013-A and UUIPGi014-A) from cases with Down syndrome and full trisomy for chromosome 21 (T21).

Aberrant splicing due to a novelRPS7variant causes Diamond-Blackfan Anemia associated with spontaneous remission and meningocele

DNA methylation changes in Down syndrome derived neural iPSCs uncover co-dysregulation of ZNF and HOX3 families of transcription factors

Generation of a human Neurochondrin deficient iPSC line KICRi002-A-3 using CRISPR/Cas9

Incontinentia pigmenti: Generation of an IKBKG deficient human iPSC line (KICRi002-A-1) on a 46,XY background using CRISPR/Cas9

Collaborators (7)

Joey Lau

Marc Friedlander

Andrey Vinogradov

xingqi chen

Bryndis Birnir

Tanja Hyvärinen

Niklas Dahl

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