Jeremy Shaw

University of Virginia

United States

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Articles (14)

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2025

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2024

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2024

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2020

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2020

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2020

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2020

Collaborators (7)

Charles E. Chalfant

University of Virginia

UNITED STATES

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Bart Tummers

King's College London

UNITED KINGDOM

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Bishal Paudel

Assistant Professor

University of Kansas Cancer Center

UNITED STATES

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Kelsey Fisher-Wellman

Associate Professor

Atrium Health Wake Forest Baptist

UNITED STATES

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Aaron Viny

Assistant Professor of Medicine

Columbia University Irving Medical Center

UNITED STATES

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Halime Kalkavan

University Hospital Essen

GERMANY

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Stephane Pelletier

Clinical Associate Professor

Indiana University School of Medicine

UNITED STATES

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Jeremy Shaw

Research Interests

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Articles (14)

FLIP L permits apoptotic and inflammatory signaling and inhibits necroptosis in mice without Caspase-8 oligomerization

Acute myeloid leukemia stratifies as 2 clinically relevant sphingolipidomic subtypes

The interaction between RIPK1 and FADD controls perinatal lethality and inflammation

Non-lethal outcomes of engaging regulated cell death pathways in cancer

Simultaneous Inhibition of Ceramide Hydrolysis and Glycosylation Synergizes to Corrupt Mitochondrial Respiration and Signal Caspase Driven Cell Death in Drug-Resistant Acute Myeloid Leukemia

Acid Ceramidase Inhibitor LCL-805 Antagonizes Akt Signaling and Promotes Iron-Dependent Cell Death in Acute Myeloid Leukemia.

Caspase-8 and FADD prevent spontaneous ZBP1 expression and necroptosis

Ceramide nanoliposomes augment the efficacy of venetoclax and cytarabine in models of acute myeloid leukemia.

Harnessing the power of sphingolipids: Prospects for acute myeloid leukemia.

IGF1R and Src inhibition induce synergistic cytotoxicity in HNSCC through inhibition of FAK.

Inhibition of Lysosomal Function Mitigates Protective Mitophagy and Augments Ceramide Nanoliposome–Induced Cell Death in Head and Neck Squamous Cell Carcinoma

Role of SPTSSB-Regulated de Novo Sphingolipid Synthesis in Prostate Cancer Depends on Androgen Receptor Signaling

Glucosylceramide production maintains colon integrity in response to Bacteroides fragilis toxin-induced colon epithelial cell signaling.

Glucosylceramide synthase maintains influenza virus entry and infection.

Collaborators (7)

Charles E. Chalfant

Bart Tummers

Bishal Paudel

Kelsey Fisher-Wellman

Aaron Viny

Halime Kalkavan

Stephane Pelletier

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