Jesús José Ferre Fernández

Assistant Professor

Universidad de Murcia Facultad de Medicina

Spain

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About

Jesús José Ferre Fernández is an Assistant Professor at Universidad de Castilla-La Mancha Facultad de Medicina, Spain. His research primarily focuses on developmental biology and ophthalmology, particularly through the use of zebrafish models. Recent articles highlight his investigations into the roles of myocilin and foxc1 genes in ocular disorders and their associated molecular mechanisms.

Articles (11)

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2022

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2022

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2022

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2021

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2020

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2020

Collaborators (3)

Jose Daniel Aroca-Aguilar

Profesor Titular de Universidad. Área de Genética

University of Castilla-La Mancha

SPAIN

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Laura Fernández-Sánchez

Associate professor of Pharmacology

Universidad de Alicante

SPAIN

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Julio Escribano

Catedrático (Full Professor)

University of Castilla-La Mancha

SPAIN

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Jesús José Ferre Fernández

Research Interests

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Articles (11)

Transgenic Overexpression of Myocilin Leads to Variable Ocular Anterior Segment and Retinal Alterations Associated with Extracellular Matrix Abnormalities in Adult Zebrafish

CRISPR-Cas9-mediated functional dissection of the foxc1 genomic region in zebrafish identifies critical conserved cis-regulatory elements

Transgenic Overexpression of Myocilin Leads to Variable Ocular Anterior Segment and Retinal Alterations Associated with Extracellular Matrix Abnormalities in Adult Zebrafish

Knockout of myoc Provides Evidence for the Role of Myocilin in Zebrafish Sex Determination Associated with Wnt Signalling Downregulation

Role of GUCA1C in Primary Congenital Glaucoma and in the Retina: Functional Evaluation in Zebrafish

CPAMD8 loss-of-function underlies non-dominant congenital glaucoma with variable anterior segment dysgenesis and abnormal extracellular matrix

Disruption of foxc1 genes in zebrafish results in dosage-dependent phenotypes overlapping Axenfeld-Rieger syndrome

Disruption of foxc1 genes in zebrafish results in dosage-dependent phenotypes overlapping Axenfeld-Rieger syndrome

Apparent Radiological Improvement in an Infant With Labrune Syndrome Treated With Bevacizumab

Apparent Radiological Improvement in an Infant With Labrune Syndrome Treated With Bevacizumab

CPAMD8 loss-of-function underlies non-dominant congenital glaucoma with variable anterior segment dysgenesis and abnormal extracellular matrix

Collaborators (3)

Jose Daniel Aroca-Aguilar

Laura Fernández-Sánchez

Julio Escribano

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