Jill Bargonetti

Professor

City University of New York Graduate School and University Center

United States

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About

Jill Bargonetti is a Professor at the City University of New York Graduate School and University Center. Her research focuses on cancer biology, specifically the roles of mutant p53, MDM2, and MDM4 in DNA repair mechanisms and tumor progression. Recent articles include investigations into the accumulation of mutant p53, its mechanisms in cancer metastasis, and the therapeutic potentials of PARP inhibitors in treatment strategies for p53-mutated tumors.

Articles (15)

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2025

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2025

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2024

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2024

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2024

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (6)

Ines Munoz

Head of Unit (Protein Crystallography Unit)

Centro Nacional de Investigaciones Oncológicas

SPAIN

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Javier Querol-Garcia

Centro de Investigaciones Biológicas Margarita Salas

SPAIN

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Galina Khramtsova

University of Chicago

UNITED STATES

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Sheryl Roberts

Assistant Professor

Wayne State University

UNITED STATES

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Zachary Samuels

The Graduate Center, CUNY

UNITED STATES

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Chun-Yuan Chang

Rutgers University

UNITED STATES

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Jill Bargonetti

Research Interests

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Articles (15)

A cancer persistent DNA repair circuit driven by MDM2, MDM4 (MDMX), and mutant p53 for recruitment of MDC1 and 53BP1 on chromatin

Lipogenic enzyme FASN promotes mutant p53 accumulation and gain-of-function through palmitoylation

Patient-derived tumor organoids with p53 mutations, and not wild-type p53, are sensitive to synergistic combination PARP inhibitor treatment

Chemokine CXCL12 Activates CXC Receptor 4 Metastasis Signaling Through the Upregulation of a CXCL12/CXCR4/MDMX (MDM4) Axis

p53 suppresses MHC class II presentation by intestinal epithelium to protect against radiation-induced gastrointestinal syndrome

The C-terminus of Gain-of-Function Mutant p53 R273H Is Required for Association with PARP1 and Poly-ADP-Ribose

Mutant C. elegans p53 Together with Gain-of-Function GLP-1/Notch Decreases UVC-Damage-Induced Germline Cell Death but Increases PARP Inhibitor-Induced Germline Cell Death

Small molecule MMRi62 targets MDM4 for degradation and induces leukemic cell apoptosis regardless of p53 status

PARP-Targeted Auger Therapy in p53 Mutant Colon Cancer Xenograft Mouse Models

Oligomerization of Mutant p53 R273H is not Required for Gain-of-Function Chromatin Associated Activities.

Frame-shift mediated reduction of gain-of-function p53 R273H and deletion of the R273H C-terminus in breast cancer cells result in replication-stress sensitivity.

Targeting Triple Negative Breast Cancer with a Nucleus-Directed p53 Tetramerization Domain Peptide

How choreostorming informs thinking in molecular genetics and cancer biology

<p>MDM2-C Functions as an E3 Ubiquitin Ligase</p>

A protein in the yeast saccharomyces cerevisiae presents dna binding homology to the p53 checkpoint protein and tumor suppressor

Collaborators (6)

Ines Munoz

Javier Querol-Garcia

Galina Khramtsova

Sheryl Roberts

Zachary Samuels

Chun-Yuan Chang

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