Joanna Mika

Prof. dr hab. n. med.

Poland

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Prof. dr hab. n. med. Joanna Mika is a faculty member at the Institute of Pharmacology, PAS in Poland. Her research primarily focuses on neuropathic pain and pharmacological modulation of immune responses, particularly the roles of various chemokines and their receptors. Recent publications include studies on the effects of astaxanthin and mirogabalin in pain management, as well as the complex interactions of immune cells in diabetic neuropathy and obesity-induced hypersensitivity.

Articles (16)

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2025

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2022

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2022

Collaborators (7)

Wioletta Makuch

Maj Institute of Pharmacology Polish Academy of Sciences

POLAND

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MAGDALENA KOCOT-KĘPSKA

adjunct PROFESSOR

Uniwersytet Jagiellonski Collegium Medicum

POLAND

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Katarzyna Ciapała

Institute of Pharmacology

POLAND

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Katarzyna Pawlik

Institute of Pharmacology

POLAND

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Zajączkowska Renata

assistant professor

Uniwersytet Jagiellonski Collegium Medicum

POLAND

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Elżbieta Karczewska

Associate professor / PhD, DSc

Uniwersytet Jagiellonski Collegium Medicum

POLAND

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Anna Maria Waszkielewicz

Jagiellonian University Medical College

POLAND

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Joanna Mika

Research Interests

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Articles (16)

Pharmacological modulation of neutrophils, in contrast to that of macrophages/microglia, is sex independent and delays the development of morphine tolerance in a mouse model of neuropathic pain

A New Application for Cenicriviroc, a Dual CCR2/CCR5 Antagonist, in the Treatment of Painful Diabetic Neuropathy in a Mouse Model

CC Chemokine Family Members’ Modulation as a Novel Approach for Treating Central Nervous System and Peripheral Nervous System Injury—A Review of Clinical and Experimental Findings

Astaxanthin has a beneficial influence on pain-related symptoms and opioid-induced hyperalgesia in mice with diabetic neuropathy-evidence from behavioral studies

A Missing Puzzle in Preclinical Studies—Are CCR2, CCR5, and Their Ligands’ Roles Similar in Obesity-Induced Hypersensitivity and Diabetic Neuropathy?—Evidence from Rodent Models and Clinical Studies

Mirogabalin Decreases Pain-like Behaviors by Inhibiting the Microglial/Macrophage Activation, p38MAPK Signaling, and Pronociceptive CCL2 and CCL5 Release in a Mouse Model of Neuropathic Pain

Phenytoin Decreases Pain-like Behaviors and Improves Opioid Analgesia in a Rat Model of Neuropathic Pain

Targeting Members of the Chemokine Family as a Novel Approach to Treating Neuropathic Pain

Analgesic Effects of Fisetin, Peimine, Astaxanthin, Artemisinin, Bardoxolone Methyl and 740 Y-P and Their Influence on Opioid Analgesia in a Mouse Model of Neuropathic Pain

Pharmacological Modulation of the MIP-1 Family and Their Receptors Reduces Neuropathic Pain Symptoms and Influences Morphine Analgesia: Evidence from a Mouse Model

Effect of pharmacological modulation of the kynurenine pathway on pain-related behavior and opioid analgesia in a mouse model of neuropathic pain

Advances in Neuropathic Pain Research: Selected Intracellular Factors as Potential Targets for Multidirectional Analgesics

Mirogabalin Decreases Pain-like Behaviours and Improves Opioid and Ketamine Antinociception in a Mouse Model of Neuropathic Pain

Pharmacological Evidence of the Important Roles of CCR1 and CCR3 and Their Endogenous Ligands CCL2/7/8 in Hypersensitivity Based on a Murine Model of Neuropathic Pain

CC Chemokine Receptor 4 (CCR4) as a Possible New Target for Therapy

Promising anticonvulsant and/or analgesic compounds among 5-chloro-2- or 5-chloro-4-methyl derivatives of xanthone coupled to aminoalkanol moieties - design, synthesis and pharmacological evaluation.

Collaborators (7)

Wioletta Makuch

MAGDALENA KOCOT-KĘPSKA

Katarzyna Ciapała

Katarzyna Pawlik

Zajączkowska Renata

Elżbieta Karczewska

Anna Maria Waszkielewicz

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