Jordan Abbott

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Associate Professor

University of Colorado Anschutz Medical Campus

United States

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About

Dr. Jordan Abbott is an Associate Professor at the University of Colorado - Anschutz Medical Campus, USA. His research areas include immunology, specifically focusing on immune disorders, gene therapy in neuromuscular diseases, and the clinical implications of immunodeficiencies. Recent publications highlight his work on the longitudinal monitoring of B cell populations, the immune status of patients with genetic deletions, and the implications of autophagy in immune dysregulation.

Recent Grants

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Circulating Unconventional T Cells in Pediatric Heart Transplant Recipients: From Pathogenesis to Biomarker

Open Date: 2022-07-01

Close Date: 2025-06-30

Articles (13)

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2025

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2020

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2020

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2020

Collaborators (5)

KUANG-CHIH HSIAO

University of Auckland

NEW ZEALAND

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Jessica Hui

Rush University Rush Medical College

UNITED STATES

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Morgan MacBeth

Senior Professional Research Assistant

University of Colorado Anschutz Medical Campus

UNITED STATES

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Despina Moshous

Imagine Institute for Genetic Diseases

FRANCE

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Laura Ann Wang

Assistant Professor

University of Colorado Anschutz Medical Campus

UNITED STATES

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Jordan Abbott

Research Interests

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About

Recent Grants

Articles (13)

Longitudinal monitoring of class-switched memory-B cell proportions identifies plausible germinal center failure in patients with suspected immune disorders.

Clinical and functional spectrum of RAC2-related immunodeficiency

Meeting Report: 2023 Muscular Dystrophy Association Summit on 'Safety and Challenges in Gene Therapy of Neuromuscular Diseases'.

The Immune Status of Patients with 16p11.2 Deletion Syndrome

The pulmonary effects of STAT3 deficiency.

Autophagy-associated immune dysregulation and hyperplasia in a patient with compound heterozygous mutations in <i>ATG9A</i>

Fluctuations in quality of life and immune responses during intravenous immunoglobulin infusion cycles.

"Common variable immunodeficiency: Challenges for diagnosis".

Registries are shaping how we think about primary immunodeficiency diseases.

Cytotoxic T-Lymphocyte-Associated Antigen 4 Haploinsufficiency Mimics Difficult-to-Treat Inflammatory Bowel Disease.

Heterozygous IKKβ activation loop mutation results in a complex immunodeficiency syndrome.

Expression and activation of the steroidogenic enzyme CYP11A1 is associated with IL-13 production in T cells from peanut allergic children.

Use of Bortezomib in the Treatment of C3 Glomerulonephritis Refractory to Eculizumab and Rituximab.

Collaborators (5)

KUANG-CHIH HSIAO

Jessica Hui

Morgan MacBeth

Despina Moshous

Laura Ann Wang

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