Joshua M. Thurman

United States

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Articles (17)

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2025

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2022

Collaborators (12)

Jost Klawitter

Assistant Professor

University of Denver

UNITED STATES

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Debbie Gipson

Professor

University of Michigan

UNITED STATES

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Eva Nozik

Professor

University of Denver

UNITED STATES

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Giovanni Camussi

Full professor of Nephrology

Università degli Studi di Torino

ITALY

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Richard Alexander Harrison

Honorary Professor

Cardiff University

UNITED KINGDOM

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Paola Pontrelli

Università degli Studi di Bari Aldo Moro Scuola di Medicina

ITALY

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Fabio Sallustio

Associate Professor

University of Bari Aldo Moro

ITALY

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Yong Guo

Assistant professor

Mayo Clinic Minnesota

UNITED STATES

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Felix Poppelaars

University of Colorado Anschutz Medical Campus

UNITED STATES

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Bradley Dixon

Associate Professor of Pediatrics

University of Colorado School of Medicine

UNITED STATES

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Howard Trachtman

Martin Shkreli Professor of Pediatric Nephrology

NYU School of Medicine

UNITED STATES

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Stefania BRUNO

Associate Professor of Laboratory Medicine

University of Turin

ITALY

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Joshua M. Thurman

Research Interests

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Articles (17)

Monitoring of Allograft Adaptation After Kidney Transplantation in Pediatric Patients by Targeted Plasma Metabolomics

Friend or foe: assessing the value of animal models for facilitating clinical breakthroughs in complement research

Chronic kidney disease enhances alternative pathway activity: a new paradigm

A Newly Identified Protective Role of C5a Receptor 1 in Kidney Tubules against Toxin-Induced Acute Kidney Injury

Activation of platelets and the complement system in mice with Schistosoma-induced pulmonary hypertension

The Complement System: An Important New Therapeutic Target in IgA Nephropathy

The complement alternative pathway in health and disease—activation or amplification?

The susceptibility of the kidney to alternative pathway activation—A hypothesis

Developing an Edema Clinician-Reported Outcome Measure for Nephrotic Syndrome

Female but Not Male Mice Deficient in Soluble IgM Are Susceptible to Chemically Induced Glomerular Injury

MOGAD patient autoantibodies induce complement, phagocytosis, and cellular cytotoxicity

The role of complement in kidney disease

Extracellular vesicles derived from patients with antibody-mediated rejection induce tubular senescence and endothelial to mesenchymal transition in renal cells

The complement system in pediatric acute kidney injury.

Factor H related proteins modulate complement activation on kidney cells

Alternative pathway diagnostics.

The Immune System and Idiopathic Nephrotic Syndrome.

Collaborators (12)

Jost Klawitter

Debbie Gipson

Eva Nozik

Giovanni Camussi

Richard Alexander Harrison

Paola Pontrelli

Fabio Sallustio

Yong Guo

Felix Poppelaars

Bradley Dixon

Howard Trachtman

Stefania BRUNO

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