JP Jin

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Professor

University of Chicago

United States

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JP Jin is a Professor at the University of Illinois at Chicago, USA. His research focuses on the molecular mechanisms of troponin and its variants in muscle function, including their roles in contractile kinetics and diseases such as TNNT1 myopathy. He has published extensively on topics such as mechanoregulation, troponin modifications, and their evolutionary implications in vertebrate cardiac function.

Recent Grants

Grant: Close

Computer modeling of myosin binding protein C and its effects on cardiac contraction

Open Date: 2019-04-01

Close Date: 2023-03-31

Grant: Close

Modification of Troponin T to Improve Cardiac Function in Heart Failure

Open Date: 2018-06-01

Close Date: 2022-02-28

Grant: Close

Rabbit model for cystic fibrosis

Open Date: 2016-07-05

Close Date: 2020-06-30

Grant: Close

Regulation of Troponin I in Cardiac Adaptation & Failure

Open Date: 2016-05-01

Close Date: 2020-03-31

Grant: Close

Detection of Host Response In Clostridium Difficile Infection

Open Date: 2015-07-01

Close Date: 2017-12-31

Articles (13)

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2025

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (1)

Masataka Kawai

Professor

Iowa State University

UNITED STATES

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JP Jin

Research Interests

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About

Recent Grants

Articles (13)

The highly conserved C-terminal end segment of troponin T binds tropomyosin and actin to function in modulating contractile kinetics

Potential cytotoxicity of truncated slow skeletal muscle troponin T (ssTnT) in a loss of function TNNT1 myopathy mouse model

Mechanoregulation and function of calponin and transgelin

N-terminal truncated cardiac troponin I enhances Frank-Starling response by increasing myofilament sensitivity to resting tension

Early Divergence of the C-Terminal Variable Region of Troponin T Via a Pair of Mutually Exclusive Alternatively Spliced Exons Followed by a Selective Fixation in Vertebrate Heart

Loss of Calponin 2 causes age‐progressive proteinuria in mice

Monoclonal Antibodies as Probes to Study Ligand-Induced Conformations of Troponin Subunits.

Truncation of the N-terminus of cardiac troponin I initiates adaptive remodeling of the myocardial proteosome via phosphorylation of mechano-sensitive signaling pathways.

Evolution of the N-Terminal Regulation of Cardiac Troponin I for Heart Function of Tetrapods: Lungfish Presents an Example of the Emergence of Novel Submolecular Structure to Lead the Capacity of Adaptation

Troponin Variants as Markers of Skeletal Muscle Health and Diseases.

A rapid degradation of calponin 2 is required for cytokinesis

Mechanisms of Frank-Starling law of the heart and stretch activation in striated muscles may have a common molecular origin

Deletion of Calponin 2 Reduces the Formation of Postoperative Peritoneal Adhesions.

Collaborators (1)

Masataka Kawai

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