juan rosado

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Professor in Physiology

Universidad de Extremadura

Spain

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Juan Rosado is a Professor in Physiology at the University of Extremadura, Spain. His research focuses on calcium channels, particularly Orai1 subunits, and their roles in various cancer cells and physiological processes. Recent articles authored by Professor Rosado delve into the mechanisms of store-operated calcium entry and its implications for breast and colorectal cancer, as well as the physiological roles of TRP channels in sleep and circadian rhythms.

Recent Grants

Grant: Close

Adquisition of a high resolution advanced microscopy system

Open Date: 2021-09-01

Close Date: 2022-12-01

Grant: Close

Estudio del papel de las variantes del canal Orai1, Orai1α y Orai1β, en la función celular

Open Date: 2021-06-01

Close Date: 2024-05-01

Grant: Close

REMODELADO DE LA EXPRESION DE STIM Y ORAI Y DE SUS MECANISMOS REGULADORES EN EL CANCER DE MAMA

Open Date: 2020-06-01

Close Date: 2023-05-01

Grant: Close

Adquisition of a system of videomicroscopy for 3D cell culture

Open Date: 2019-11-01

Close Date:

Grant: Close

INTERACTION OF CAPACITATIVE CALCIUM CHANNELS, ORAI AND TRPC, THEIR FEATURES AND RELEVANCE IN THE PATHOPHYSIOLOGY OF BREAST CANCER

Open Date: 2017-06-01

Close Date: 2020-05-01

Articles (20)

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Universidad de Extremadura

SPAIN

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juan rosado

Research Interests

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Recent Grants

Articles (20)

A subset of Orai1α and Orai1β subunits heteromerizes to form CRAC channels.

Filamin A C-terminal fragment modulates Orai1 expression by inhibition of protein degradation

Feedback modulation of Orai1α and Orai1β protein content mediated by STIM proteins

Inhibition of adenylyl cyclase 8 prevents the upregulation of Orai1 channel, which improves cardiac function after myocardial infarction

Postbiotics of Lacticaseibacillus paracasei CECT 9610 and Lactiplantibacillus plantarum CECT 9608 attenuates store-operated calcium entry and FAK phosphorylation in colorectal cancer cells

Differential functional role of Orai1 variants in constitutive Ca2+ entry and calcification in luminal breast cancer cells

Extended Synaptotagmins 1 and 2 Are Required for Store-Operated Calcium Entry, Cell Migration and Viability in Breast Cancer Cells

The physiological role of TRP channels in sleep and circadian rhythm

Reduced Ca2+ mobilization in neonatal human platelets involves SARAF and pannexin‐1

Functional differences in agonist‐induced plasma membrane expression of Orai1α and Orai1β

New Insights into the Reparative Angiogenesis after Myocardial Infarction

The Ca<sup>2+</sup> Sensor STIM in Human Diseases

Secretion of Interleukin 6 in Human Skeletal Muscle Cultures Depends on Ca2+ Signalling

Role of Orai‐family channels in the activation and regulation of transcriptional activity

Correction: Sanchez-Collado et al. Orai2 Modulates Store-Operated Ca2+ Entry and Cell Cycle Progression in Breast Cancer Cells. Cancers 2021, 14, 114

The store-operated Ca2+ channel Orai1α is required for agonist-evoked NF-κB activation by a mechanism dependent on PKCβ2

Orai1α and Orai1β support calcium entry and mammosphere formation in breast cancer stem cells

Store-Operated Calcium Entry in Breast Cancer Cells Is Insensitive to Orai1 and STIM1 N-Linked Glycosylation

Similarities and Differences between the Orai1 Variants: Orai1α and Orai1β

PKC-Mediated Orai1 Channel Phosphorylation Modulates Ca2+ Signaling in HeLa Cells

Collaborators (5)

WALDO LUIS GARCIA-JIMÉNEZ

Tarik Smani Hajami

PEDRO C REDONDO

PEDRO J CAMELLO

Francisco Javier Martin-Romero

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