Jun-Li Liu

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Associate Professor

McGill University

Canada

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About

Jun-Li Liu is an Associate Professor at McGill University in Canada. His research primarily focuses on the mechanisms involved in β-cell growth and insulin resistance, the differentiation of functional β-cells from stem cells, and the roles of cellular communication network factors in metabolic disorders. He has also explored the impact of sex hormones on cardiometabolic health and their implications for tissue repair and cancer development.

Recent Grants

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CCN5/WISP2

Open Date: 2021-01-01

Close Date: 2026-01-01

Articles (14)

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2025

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2021

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2021

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2020

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Jun-Li Liu

Research Interests

Explore related searches

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About

Recent Grants

Articles (14)

Endogenous Expression of Cellular Communication Network Factor 5 (CCN5/WISP2) on Normal β-Cells Growth and Insulin Resistance Caused by Diet-Induced Obesity

A stepwise approach to deriving functional β-cells from human embryonic or induced pluripotent stem cells

Controversial Roles of Regenerating Family Proteins in Tissue Repair and Tumor Development

Beyond reproduction: unraveling the impact of sex hormones on cardiometabolic health

Penetration enhancers strengthen tough hydrogel bioadhesion and modulate locoregional drug delivery

Recent progress on the role of cellular communication network factors (CCN) 3, 4 and 6 in regulating adiposity, liver fibrosis and pancreatic islets

Recombinant Reg3α Prevents Islet β-Cell Apoptosis and Promotes β-Cell Regeneration

An integrated model of acinar to ductal metaplasia-related N7-methyladenosine regulators predicts prognosis and immunotherapy in pancreatic carcinoma based on digital spatial profiling

An integrated model of acinar to ductal metaplasia-related N7-methyladenosine regulators predicts prognosis and immunotherapy in pancreatic carcinoma based on digital spatial profiling

Metabolic Effects of CCN5/WISP2 Gene Deficiency and Transgenic Overexpression in Mice

Metabolic Effects of CCN5/WISP2 Gene Deficiency and Transgenic Overexpression in Mice

REG3A/REG3B promotes acinar to ductal metaplasia through binding to EXTL3 and activating the RAS-RAF-MEK-ERK signaling pathway

REG3A/REG3B promotes acinar to ductal metaplasia through binding to EXTL3 and activating the RAS-RAF-MEK-ERK signaling pathway

Controversial Roles of Gut Microbiota-Derived Short-Chain Fatty Acids (SCFAs) on Pancreatic beta-Cell Growth and Insulin Secretion

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