Kazuhisa Nakayama

教授 / Professor

Kyoto University

Japan

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Articles (14)

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2022

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2021

Collaborators (4)

Yohei Katoh

Assistant Professor

Kyoto University

JAPAN

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Hye-Won Shin

Kyoto University

JAPAN

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Shuhei Chiba

Assistant Professor

Tohoku University

JAPAN

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David Stephens

University of Bristol

UNITED KINGDOM

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Kazuhisa Nakayama

Research Interests

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Articles (14)

Novel phosphatidylinositol flippases contribute to phosphoinositide homeostasis in the plasma membrane

Defects in diffusion barrier function of ciliary transition zone caused by ciliopathy variations of TMEM218

Glucosylceramide flippases contribute to cellular glucosylceramide homeostasis

Lysosomal membrane integrity in fibroblasts derived from patients with Gaucher disease

Multiple interactions of the dynein-2 complex with the IFT-B complex are required for effective intraflagellar transport

Compound heterozygous IFT81 variations in a skeletal ciliopathy patient cause Bardet–Biedl syndrome-like ciliary defects

Dynein-2–driven intraciliary retrograde trafficking indirectly requires multiple interactions of IFT54 in the IFT-B complex with the dynein-2 complex

Disease-associated mutations in WDR34 lead to diverse impacts on the assembly and function of dynein-2

CEP19–RABL2–IFT-B axis controls BBSome-mediated ciliary GPCR export

BROMI/TBC1D32 together with CCRK/CDK20 and FAM149B1/JBTS36 contributes to intraflagellar transport turnaround involving ICK/CILK1

Molecular basis underlying the ciliary defects caused by IFT52 variations found in skeletal ciliopathies

Impaired cooperation between IFT74/BBS22–IFT81 and IFT25–IFT27/BBS19 causes Bardet-Biedl syndrome

Combinations of deletion and missense variations of the dynein-2 DYNC2LI1 subunit found in skeletal ciliopathies cause ciliary defects

CCRK/CDK20 regulates ciliary retrograde protein trafficking via interacting with BROMI/TBC1D32

Collaborators (4)

Yohei Katoh

Hye-Won Shin

Shuhei Chiba

David Stephens

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