Kei IIDA

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Lecturer

Kindai University

Japan

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About

Dr. Kei Iida is a Lecturer at Kindai University in Japan. His research focuses on genetic splicing mechanisms and their implications in various diseases, as evidenced by his recent publications on RNA mis-splicing in cardiac Fabry disease, COVID-19 vulnerability, and therapeutic strategies for cancer. Dr. Iida's work also explores innovative in vitro models related to liver disease and important cell interactions within metastatic contexts.

Recent Grants

Grant: Close

Mechanisms for the initial process of hair cell regeneration in avian inner ears

Open Date: 2020-04-01

Close Date: 2023-03-31

Grant: Close

Elucidation of splicing-code with RNA-specialized machine learning system toward overcoming hereditary diseases having splicing misregulations

Open Date: 2020-04-01

Close Date: 2023-03-31

Grant: Close

Deciphering neuroepigenetics that regulate the neural differentiation

Open Date: 2019-04-01

Close Date: 2022-03-31

Grant: Close

Widely spread RNA modification at neuronal synapses add functional diversity to local transcriptome

Open Date: 2017-04-01

Close Date: 2020-03-31

Grant: Close

CRISPRによるRNA病モデルｉＰＳ細胞・動物の構築と病態解明・治療薬創製

Open Date: 2015-05-29

Close Date: 2020-03-31

Articles (13)

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2025

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2025

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2021

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JAPAN

Assistant Professor

Kyoto University

JAPAN

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Kei IIDA

Research Interests

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Contact this professor

About

Recent Grants

Articles (13)

Invention of an oral medication for cardiac Fabry disease caused by RNA mis-splicing

Switching of OAS1 splicing isoforms overcomes SNP-derived vulnerability to SARS-CoV-2 infection

Integrated-gut-liver-on-a-chip platform as an in vitro human model of non-alcoholic fatty liver disease

Secretory GFP reconstitution labeling of neighboring cells interrogates cell–cell interactions in metastatic niches

Method for Identifying Sequence Motifs in Pre-miRNAs for Small-Molecule Binding

Chemical induction of splice-neoantigens attenuates tumor growth in a preclinical model of colorectal cancer

Author Correction: Therapeutic manipulation of IKBKAP mis-splicing with a small molecule to cure familial dysautonomia

Therapeutic manipulation of IKBKAP mis-splicing with a small molecule to cure familial dysautonomia

CDC2-like (CLK) protein kinase inhibition as a novel targeted therapeutic strategy in prostate cancer.

S1PR3-G<sub>12</sub>-biased agonist ALESIA targets cancer metabolism and promotes glucose starvation.

Improvement of Moloney murine leukemia virus reverse transcriptase thermostability by introducing a disulfide bridge in the ribonuclease H region.

Multilateral Bioinformatics Analyses Reveal the Function-Oriented Target Specificities and Recognition of the RNA-Binding Protein SFPQ

Identification of Qk as a Glial Precursor Cell Marker that Governs the Fate Specification of Neural Stem Cells to a Glial Cell Lineage.

Collaborators (10)

Kazuhiko Nakatani

Akihide Takeuchi

Asako Murata

Toshiyuki Tsuchiya

Yoshikazu Hirai

Takahiro Kuchimaru

Ken-ichiro Kamei

Tomonari Awaya

Sebastian Oltean

Dohyun Im

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