Kenia Nunes

Associate Professor

Florida Institute of Technology

United States

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Dr. Kenia Nunes is an Associate Professor at the Florida Institute of Technology, USA. Her research focuses on vascular function, diabetes, and the effects of pharmaceuticals on biological systems, as evidenced by her recent articles. Key topics include the relationship between histological staining and corneal stiffness, the impact of HSP70 on vascular responses, and the environmental toxicity of certain antiretrovirals.

Articles (15)

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2025

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2025

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2025

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2022

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2020

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2020

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2020

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2020

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2020

Collaborators (1)

linxia gu

Florida Institute of Technology

UNITED STATES

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Kenia Nunes

Research Interests

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Articles (15)

Spatial relationship between histological staining intensity and corneal stiffness variations: Insights from AFM indentation in infant African green monkeys

Blockade of HSP70 Improves Vascular Function in a Mouse Model of Type 2 Diabetes

Toxicity of the Antiretrovirals Tenofovir Disoproxil Fumarate, Lamivudine, and Dolutegravir on Cyanobacterium Microcystis novacekii

Damage-Induced Softening of the Sclera: A Pseudo-Elastic Modeling Approach

Removal of the Active Pharmaceutical Substance Entecavir from Water via the Fenton Reaction or Action by the Cyanobacterium Microcystis novacekii

Age-Related Decline in Vascular Responses to Phenylephrine Is Associated with Reduced Levels of HSP70

Drugs from Animal Venoms: Overcoming the Challenges to Treat Erectile Dysfunction

COVID-19 and ROS Storm: What is the Forecast for Hypertension

Hypertension and Erectile Dysfunction: Breaking Down the Challenges

Dissecting the interaction between HSP70 and vascular contraction: role of [Formula: see text] handling mechanisms.

New insights into RhoA/Rho-kinase signaling: a key regulator of vascular contraction.

Blockade of the TLR4-MD2 complex lowers blood pressure and improves vascular function in a murine model of type 1 diabetes.

An additional physiological role for HSP70: Assistance of vascular reactivity.

Blockade of Toll-like receptor 4 (TLR4) reduces oxidative stress and restores phospho-ERK1/2 levels in Leydig cells exposed to high glucose.

Angiotensin (1-7) Inhibits Ang II-mediated ERK1/2 Activation by Stimulating MKP-1 Activation in Vascular Smooth Muscle Cells.

Collaborators (1)

linxia gu

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