Kenneth Humphries

Associate Professor

Oklahoma Medical Research Foundation

United States

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Dr. Kenneth Humphries is an Associate Professor at the Oklahoma Medical Research Foundation in the United States. His research areas encompass cardiac metabolism, mitochondrial function, and the effects of various inhibitors on metabolic pathways. Recent publications highlight his work on targeted drug delivery systems for myocardial infarction, as well as the regulation of metabolic processes in both cardiac and adipose tissues.

Articles (12)

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2025

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2025

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2025

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2024

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2023

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2023

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (4)

Maria Fernanda Mendez Garcia

Assistant Professor of Research

University of Oklahoma Health Sciences Center

UNITED STATES

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Dawid Makosa

University of Western Australia

AUSTRALIA

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Wan Hee Yoon

Associate Professor

Oklahoma Medical Research Foundation

UNITED STATES

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Ann Louise Olson

Professor

University of Oklahoma College of Medicine

UNITED STATES

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Kenneth Humphries

Research Interests

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Articles (12)

Cardiotropic ligand-guided liposomes for myocardial delivery of atorvastatin in a mouse model of myocardial infarction

The Identification of a Key Regulator of Mitochondrial Metabolism, the LRPPRC Protein, as a Novel Therapeutic Target in SDHA-Overexpressing Ovarian Tumors

Mechanistic studies of PFKFB2 reveal a novel inhibitor of its kinase activity

Effect of Low-Level Tragus Stimulation on Cardiac Metabolism in Heart Failure with Preserved Ejection Fraction: A Transcriptomics-Based Analysis

The loss of cardiac SIRT3 decreases metabolic flexibility and proteostasis in an age-dependent manner

Increased cardiac PFK-2 protects against high-fat diet-induced cardiomyopathy and mediates beneficial systemic metabolic effects.

Insulin signaling alters antioxidant capacity in the diabetic heart

Sirt5 Deficiency Causes Posttranslational Protein Malonylation and Dysregulated Cellular Metabolism in Chondrocytes Under Obesity Conditions.

PFKFB3-dependent glucose metabolism regulates 3T3-L1 adipocyte development.

Diabetes induced decreases in PKA signaling in cardiomyocytes: The role of insulin

A biallelic pathogenic variant in the OGDH gene results in a neurological disorder with features of a mitochondrial disease.

Recent advances in understanding glucose transport and glucose disposal.

Collaborators (4)

Maria Fernanda Mendez Garcia

Dawid Makosa

Wan Hee Yoon

Ann Louise Olson

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