Klaus E. Schmitz-Abe

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Associate Professor

University of Miami

United States

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About

Klaus E. Schmitz-Abe is an Associate Professor at the University of Miami, USA. His research areas encompass immunology, genetics, and disease modeling, focusing on topics such as Long COVID, neurodevelopmental syndromes, and the microbiome's role in oral tolerance. He has contributed to the understanding of candidate gene variants and their implications in various health conditions, including cystic fibrosis and hyper-IgE syndrome.

Recent Grants

Grant: Close

VIGOR: Virtual Genome Center for Infant Health

Open Date: 2021-01-01

Close Date: 2026-01-01

Grant: Close

Targeting Microbial Dysbiosis in Food Allergy to Restore Tolerance

Open Date: 2021-01-01

Close Date: 2026-01-01

Grant: Close

Center for Integrated Approaches to Undiagnosed Diseases

Open Date: 2019-01-01

Close Date: 2022-01-01

Grant: Close

Control of Regulatory T Cell Homeostasis and Function by Notch Signaling

Open Date: 2017-01-01

Close Date: 2022-01-01

Grant: Close

Translational Studies of Inherited Marrow Failure and Myelodysplastic Syndromes

Open Date: 2013-01-01

Close Date: 2018-01-01

Articles (20)

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2021

Collaborators (8)

Ignazia Prigione

Istituto Giannina Gaslini

ITALY

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Arumugam R. Jayakumar

University of Miami Miller School of Medicine

UNITED STATES

Assistant Professor

Boston Children's Hospital

UNITED STATES

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Klaus E. Schmitz-Abe

Research Interests

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About

Recent Grants

Articles (20)

Persistent Inflammation, Maladaptive Remodeling, and Fibrosis in the Kidney Following Long COVID-like MHV-1 Mouse Model

Recessive variants in WSB2 encoding a substrate receptor of E3 ubiquitin ligase underlie a neurodevelopmental syndrome

Unique Signatures of Highly Constrained Genes Across Publicly Available Genomic Databases

RELMβ sets the threshold for microbiome-dependent oral tolerance

High number of candidate gene variants are identified as disease‐causing in a period of 4 years

DOCK8 deficiency due to a deep intronic variant in two kindreds with hyper-IgE syndrome

Immunological biomarkers associated with survival in a cohort of Argentinian patients with common variable immunodeficiency

The solute carrier family 26 member 9 modifies rapidly progressing cystic fibrosis associated with homozygous F508del CFTR mutation

Integrated multi‐omics approach reveals the role of striated muscle preferentially expressed protein kinase in skeletal muscle including its relationship with myospryn complex

An intronic variant in TBX4 in a single family with variable and severe pulmonary manifestations

Severe allergic dysregulation due to a gain of function mutation in the transcription factor STAT6

Reanalysis of clinical exome identifies the second variant in two individuals with recessive disorders

The Notch1/CD22 signaling axis disrupts Treg function in SARS-CoV-2–associated multisystem inflammatory syndrome in children

The Notch1/CD22 signaling axis disrupts Treg function in SARS-CoV-2–associated multisystem inflammatory syndrome in children

Atypical Localization of Eczema Discriminates DOCK8 or STAT3 Deficiencies from Atopic Dermatitis

A Stk4-Foxp3–NF-κB p65 transcriptional complex promotes T reg cell activation and homeostasis

Primary Upper Extremity Lymphedema Caused by a CELSR1 Variant

Congenital X‐linked neutropenia with myelodysplasia and somatic tetraploidy due to a germline mutation in SEPT6

Reanalysis of Exome Data Identifies Novel SLC25A46 Variants Associated with Leigh Syndrome.

Evolution and long-term outcomes of combined immunodeficiency due to CARMIL2 deficiency.

Collaborators (8)

Ignazia Prigione

Arumugam R. Jayakumar

Sara Signa

Qifei Li

Akiko Shimamura

Alan Beggs

Peggy Lai

Catherine Brownstein

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