Koichi Furukawa

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Professor

Chubu University

Japan

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About

Professor Koichi Furukawa is a faculty member at Chubu University, Japan. His research primarily focuses on the roles of extracellular vesicles and gangliosides in cancer therapeutics and bone metabolism. Recent publications highlight his work on the signaling mechanisms of exosomes in glioma cells, the impact of glycosylation on osteoblast regulation, and the functional distinctions of extracellular vesicles.

Recent Grants

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Integration of structure and molecular function in the regulation of signaling with glycosphingolipids

Open Date: 2015-04-01

Close Date: 2018-03-01

Articles (12)

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2025

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2025

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2024

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2020

Collaborators (6)

Takuma Sato

Aichi Gakuin University

JAPAN

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Keiko Kano

Nagoya University

JAPAN

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Tetsuya Okajima

Nagoya University Graduate School of Medicine

JAPAN

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Hiroshi Shiku

JAPAN

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Emi Mishiro-Sato

Center chief, Designated Lecturer

Nagoya University

JAPAN

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Takanori Ichiki

Professor

University of Tokyo

JAPAN

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Koichi Furukawa

Research Interests

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About

Recent Grants

Articles (12)

Extracellular vesicles adhere to cells primarily by interactions of integrins and GM1 with laminin

GD2 is a Crucial Ganglioside in the Signal Modulation and Application as a Target of Cancer Therapeutics

Regulation of Glycosylation in Bone Metabolism

Globo-series Gb4 activates ERK and promotes the proliferation of osteoblasts

Action Mechanisms of Exosomes Derived from GD3/GD2-Positive Glioma Cells in the Regulation of Phenotypes and Intracellular Signaling: Roles of Integrins

Disordered testosterone transport in mice lacking the ganglioside GM2/GD2 synthase gene

Attenuation of Bone Formation through a Decrease in Osteoblasts in Mutant Mice Lacking the GM2/GD2 Synthase Gene

Signaling domains of cancer-associated glycolipids

Distinguishing functional exosomes and other extracellular vesicles as a nucleic acid cargo by the anion‐exchange method

Ganglioside GD2 Enhances the Malignant Phenotypes of Melanoma Cells by Cooperating with Integrins

A nontoxigenic form of Shiga toxin 2 suppresses the production of amyloid β by altering the intracellular transport of amyloid precursor protein through its receptor-binding B-subunit

Novel Molecular Mechanisms of Gangliosides in the Nervous System Elucidated by Genetic Engineering

Collaborators (6)

Takuma Sato

Keiko Kano

Tetsuya Okajima

Hiroshi Shiku

Emi Mishiro-Sato

Takanori Ichiki

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