Launch week offerEnds in00h00m00s

annual Promonthly Pro

Kryslaine Radomski

Research Assistant Professor

Uniformed Services University of the Health Sciences F Edward Hébert School of Medicine

United States

Research Interests

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Explore related searches

Professors in United States Professors in Neuropsychology Neuropsychology professors in United States

About

Dr. Kryslaine Radomski is a Research Assistant Professor at the Uniformed Services University of the Health Sciences F Edward Hébert School of Medicine in the United States. Her research primarily focuses on traumatic brain injury, emphasizing areas such as the effects of chronic stress, axon damage, and therapeutic interventions. Recent publications include studies on the implications of repetitive mild brain injuries and the mechanisms of axon degeneration.

Articles (7)

Year:

2026

Year:

2026

Year:

2026

Year:

2023

Year:

2022

Year:

2021

Year:

2021

Collaborators (3)

Regina C. Armstrong

Professor

Uniformed Services University of the Health Sciences

UNITED STATES

View details

Pedro Brugarolas

Assistant Professor

Massachusetts General Hospital

UNITED STATES

View details

Alexandru Korotcov

Senior MRI Scientist / Assistant Professor

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Kryslaine Radomski

Research Interests

Make your next move.

Explore related searches

About

Articles (7)

Disease-modifying effects on chronic axon damage and white matter degeneration with 4-aminopyridine after acute traumatic brain injury.

Imaging Traumatic Brain Injuries in Mice with Potassium Channel PET Tracer [(18)F]3F4AP

Mapping the secondary response to traumatic brain injury using spatial transcriptomics shows acute 4-aminopyridine treatment mitigates axonal and molecular pathology.

Persistent hypersomnia following repetitive mild experimental traumatic brain injury: Roles of chronic stress and sex differences.

Acute axon damage and demyelination are mitigated by 4-aminopyridine (4-AP) therapy after experimental traumatic brain injury.

Genetic inactivation of SARM1 axon degeneration pathway improves outcome trajectory after experimental traumatic brain injury based on pathological, radiological, and functional measures.

Repetitive Blast Exposure Produces White Matter Axon Damage without Subsequent Myelin Remodeling: In Vivo Analysis of Brain Injury Using Fluorescent Reporter Mice.

Collaborators (3)

Regina C. Armstrong

Pedro Brugarolas

Alexandru Korotcov

Make your next move.