Kui Li

Professor

University of Tennessee Health Science Center

United States

Research Interests

Explore related searches

Professors in United States Professors in Immunology Immunology professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Professor Kui Li is a faculty member at the University of Tennessee Health Science Center in the United States. His research focuses on the roles of TRIM proteins in antiviral immunity, the genetic factors influencing responses to infectious diseases, and the application of large language models in health education. Recent publications include studies on innate immune responses and the impact of genetic backgrounds on disease susceptibility.

Articles (11)

Year:

2025

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2023

Year:

2022

Year:

2021

Year:

2021

Year:

Assistant Professor

University of Tennessee Health Science Center College of Medicine Memphis

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Kui Li

Research Interests

Explore related searches

Contact this professor

About

Articles (11)

Assessing the Role of Large Language Models Between ChatGPT and DeepSeek in Asthma Education for Bilingual Individuals: Comparative Study

Emerging Roles of TRIM56 in Antiviral Innate Immunity

Key roles for phosphorylation and the Coiled-coil domain in TRIM56-mediated positive regulation of TLR3-TRIF–dependent innate immunity

Deficiency of interleukin-1 receptor antagonist in mice differentially affects bone properties under different genomic backgrounds

Systems genetics of influenza A virus-infected mice identifies TRIM21 as a critical regulator of pulmonary innate immune response

Targeting intracellular Neu1 for coronavirus infection treatment

Impaired Antiviral Responses to Extracellular Double-Stranded RNA and Cytosolic DNA, but Not to Interferon-α Stimulation, in TRIM56-Deficient Cells

Ace2 and Tmprss2 Expressions Are Regulated by Dhx32 and Influence the Gastrointestinal Symptoms Caused by SARS-CoV-2

Differing susceptibility of C57BL/6J and DBA/2J mice—parents of the murine BXD family, to severe acute respiratory syndrome coronavirus infection

Genetic Dissection of the Regulatory Mechanisms of Ace2 in the Infected Mouse Lung

Influenza A virus directly modulates mouse eosinophil responses.

Collaborators (6)

Yan Cui

Benjamin M Liu

Tony Marion

Xue-Long Sun

Athena Starlard-Davenport

Heather Smallwood

Make your next move.