Kyung Lee

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Associate Professor

Icahn School of Medicine at Mount Sinai

United States

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About

Kyung Lee is an Associate Professor at the Icahn School of Medicine at Mount Sinai, United States. His research focuses on various aspects of kidney disease, including glomerular endothelial cell injury, kidney transplant failure, and the modulation of TGF-β signaling. He has authored numerous articles addressing diabetes-induced kidney complications and novel therapeutic approaches for conditions such as diabetic kidney disease and autosomal dominant polycystic kidney disease.

Recent Grants

Grant: Open

The role of Vpr-mediated cell cycle dysregulation in HIV-associated kidney disease

Open Date: 2022-08-15

Close Date: 2027-07-31

Grant: Close

Role of RARRES1 in diabetic kidney disease

Open Date: 2021-08-04

Close Date: 2025-06-30

Grant: Close

The Role of LRG1 in Diabetic Kidney Disease

Open Date: 2018-08-01

Close Date: 2023-05-31

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2022

Collaborators (3)

Luca Gusella

Associate Professor

Icahn School of Medicine at Mount Sinai

UNITED STATES

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Ye Feng

Assistant Professor

Icahn School of Medicine at Mount Sinai

UNITED STATES

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jia fu

assistant professor

Icahn School of Medicine at Mount Sinai

UNITED STATES

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Kyung Lee

Research Interests

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About

Recent Grants

Articles (25)

Targeting Krüppel-Like Factor 2 as a Novel Therapy for Glomerular Endothelial Cell Injury in Diabetic Kidney Disease

LILRB3 genetic variation is associated with kidney transplant failure in African American recipients

Modulation of TGF-β signaling new approaches toward kidney disease and fibrosis therapy

Podocyte-derived soluble RARRES1 drives kidney disease progression through direct podocyte and proximal tubular injury

LRG1 loss effectively restrains glomerular TGF-β signaling to attenuate diabetic kidney disease

Krüppel-like factor 2 is an endoprotective transcription factor in diabetic kidney disease

Role and Mechanisms of Tyro3 in Podocyte Biology and Glomerular Disease

Small Molecule Allosteric Inhibitor of HIPK2 as a Novel Therapy against Kidney Fibrosis

cGAS Activation Accelerates the Progression of Autosomal Dominant Polycystic Kidney Disease

HIPK2 C-terminal domain inhibits NF-κB signaling and renal inflammation in kidney injury

Injury in nonaged podocytes as an accelerator of glomerular aging

Cholesterol 25-Hydroxylase Protects Against Diabetic Kidney Disease by Regulating ADP Ribosylation Factor 4

RTN1A mediates diabetes-induced AKI-to-CKD transition

Cholesterol 25-Hydroxylase Protects Against Diabetic Kidney Disease by Regulating ADP Ribosylation Factor 4

TYRO3 agonist as therapy for glomerular disease

HCK induces macrophage activation to promote renal inflammation and fibrosis via suppression of autophagy

SARS-CoV-2 viral protein ORF3A injures renal tubules by interacting with TRIM59 to induce STAT3 activation

Tubular-specific expression of HIV protein Vpr leads to severe tubulointerstitial damage accompanied by progressive fibrosis and cystic development

Chronic kidney disease in a murine model of non-alcoholic steatohepatitis (NASH).

Hepatic and proximal tubule angiotensinogen play distinct roles in kidney dysfunction, glomerular and tubular injury, and fibrosis progression

Reticulon-1A mediates diabetic kidney disease progression through endoplasmic reticulum-mitochondrial contacts in tubular epithelial cells

Modulation of transforming growth factor-β-induced kidney fibrosis by leucine-rich ⍺-2 glycoprotein-1

Activation of STAT3 signaling pathway in the kidney of COVID-19 patients

Digital Spatial Profiling of Individual Glomeruli From Patients With Anti-Neutrophil Cytoplasmic Autoantibody-Associated Glomerulonephritis

HIPK2 directs cell type-specific regulation of STAT3 transcriptional activity in Th17 cell differentiation

Collaborators (3)

Luca Gusella

Ye Feng

jia fu

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