Leah Siskind

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Professor and Associate Vice-Chair

University of Louisville

United States

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About

Leah Siskind is a Professor and Associate Vice-Chair at the University of Louisville in the United States. Her research focuses on nephrology, specifically investigating the link between cancer and kidney injury, as well as the metabolic programming of immune cells in cancer progression. Recent publications include studies on kidney injury related to lung cancer and the role of ceramide synthases in aging and disease.

Recent Grants

Grant: Close

The role of neutral ceramidase in acute kidney injury and its progression to chronic kidney disease.

Open Date: 2020-01-01

Close Date: 2023-12-31

Grant: Close

Role of Ceramide-induced Kidney Cell Death in Acute Kidney Injury

Open Date: 2012-09-17

Close Date: 2018-04-30

Grant: Close

Role of Ceramide-induced Kidney Cell Death in Acute Kidney Injury

Open Date: 2012-09-17

Close Date: 2018-04-30

Grant: Close

Regulation of kidney glycosphingolipid metabolism by caloric restriction during aging

Open Date: 2011-07-01

Close Date: 2012-06-30

Grant: Close

COBRE in Lipidomics and Pathobiology

Open Date: 2002-09-26

Close Date: 2012-07-18

Articles (15)

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2022

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2020

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UNITED STATES

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Leah Siskind

Research Interests

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About

Recent Grants

Articles (15)

Introduction: The 2025 American Society of Nephrology Third Acute Kidney Injury: Bench to Bedside Conference

Remote organ cancer induces kidney injury, inflammation, and fibrosis and adversely alters renal function

Neutral ceramidase regulates breast cancer progression by metabolic programming of TREM2-associated macrophages

Expression of Ceramide Synthases in Mice and Their Roles in Regulating Acyl-Chain Sphingolipids: A Framework for Baseline Levels and Future Implications in Aging and Disease

Lung cancer-kidney cross talk induces kidney injury, interstitial fibrosis, and enhances cisplatin-induced nephrotoxicity

Modeling Cisplatin-Induced Kidney Injury to Increase Translational Potential

Inducing trained immunity in pro-metastatic macrophages to control tumor metastasis

Mutant and curli-producing E. coli enhance the disease phenotype in a hSOD1-G93A mouse model of ALS

Pharmacological inhibitors of autophagy have opposite effects in acute and chronic cisplatin-induced kidney injury

F4/80hi Resident Macrophages Contribute to Cisplatin-Induced Renal Fibrosis

Towards a molecular understanding of the overlapping and distinct roles of UBQLN1 and UBQLN2 in lung cancer progression and metastasis

Lung Cancer Induces Kidney Fibrosis and Primes the Kidney for Cisplatin-Induced Nephrotoxicity

UBR-box containing protein, UBR5, is over-expressed in human lung adenocarcinoma and is a potential therapeutic target.

C57BL/6 mice require a higher dose of cisplatin to induce renal fibrosis and CCL2 correlates with cisplatin-induced kidney injury.

Ubiquilin proteins regulate EGFR levels and activity in lung adenocarcinoma cells.

Collaborators (5)

Yi Tan

Robert Paul Friedland

Richa Singhal

Yan Yu

Robert Mitchell

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