Leila Rouhi, M.D., Ph.D.

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The University of Texas Health Science Center at Houston

United States

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The Role of Fibroblasts in the Pathogenesis of Dilated Cardiomyopathy Caused by LaminA/C Deficiency

Open Date: 2023-04-01

Close Date: 2026-03-31

Articles (6)

Year:

2021

Year:

2021

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Collaborators (3)

Ali J Marian

The University of Texas Health Science Center at Houston

UNITED STATES

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Juan Gimeno Blanes

Associate Professor

Universidad de Murcia

SPAIN

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Priyatansh Gurha

Assistant Professor

The University of Texas Health Science Center at Houston

UNITED STATES

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Leila Rouhi, M.D., Ph.D.

Research Interests

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Contact this professor

Recent Grants

Articles (6)

The EP300/TP53 pathway, a suppressor of the Hippo and canonical WNT pathways, is activated in human hearts with arrhythmogenic cardiomyopathy in the absence of overt heart failure.

Pharmacological suppression of the WNT signaling pathway attenuates age-dependent expression of the phenotype in a mouse model of arrhythmogenic cardiomyopathy.

Editors' Preamble to <i>The Journal of Cardiovascular Aging</i>.

Single Cell RNA-Sequencing Uncovers Paracrine Functions of the Epicardial-Derived Cells in Arrhythmogenic Cardiomyopathy.

Haplo-insufficiency of Tmem43 in cardiac myocytes activates the DNA damage response pathway leading to a Late-Onset Senescence-Associated pro-fibrotic cardiomyopathy.

BET bromodomain inhibition attenuates cardiac phenotype in myocyte-specific lamin A/C-deficient mice.

Collaborators (3)

Ali J Marian

Juan Gimeno Blanes

Priyatansh Gurha

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