Li Ma

Professor

The University of Texas MD Anderson Cancer Center

United States

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About

Li Ma is a Professor at The University of Texas MD Anderson Cancer Center in the United States. His research focuses on cancer biology, particularly in areas such as bone metastasis, immunotherapy, and metabolic reprogramming in the tumor microenvironment. Recent publications include investigations into the role of chromatin dynamics in cancer progression and the interplay between immune responses and tumor biology.

Articles (20)

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2025

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2021

Collaborators (7)

Alexandre Gaspar-Maia

Assistant Professor

Mayo Clinic

UNITED STATES

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Di Zhao

Assistant Professor

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Yutong Sun

Associate Professor

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Nathalie Droin

Inserm u1183

FRANCE

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Hao Zhu

Associate Professor

UNITED STATES

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Linghua Wang

Associate Professor

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Shinichi Nakagawa

JAPAN

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Li Ma

Research Interests

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About

Articles (20)

Sickle cell disease induces chromatin introversion and ferroptosis in CD8<sup>+</sup> T cells to suppress anti-tumor immunity.

In vivo CRISPR activation screen identifies acyl-CoA-binding protein as a driver of bone metastasis.

Histone methyltransferase ASH1L primes metastases and metabolic reprogramming of macrophages in the bone niche.

Long noncoding RNA Malat1 protects against osteoporosis and bone metastasis

RNA Shielding of p65 Is Required to Potentiate Oncogenic Inflammation in TET2-Mutated Clonal Hematopoiesis.

Barriers and opportunities in pancreatic cancer immunotherapy

LIFR regulates cholesterol-driven bidirectional hepatocyte-neutrophil cross-talk to promote liver regeneration.

DePARylation is critical for S phase progression and cell survival

A milestone in epithelial–mesenchymal transition

Role of Glucose Metabolic Reprogramming in Breast Cancer Progression and Drug Resistance.

New Insights into YAP/TAZ-TEAD-Mediated Gene Regulation and Biological Processes in Cancer

ZRANB1 is an NBS1 deubiquitinase and a potential target to overcome radioresistance and PARP inhibitor resistance in triple-negative breast cancer.

Immunotherapeutic Approaches for Treating Hepatocellular Carcinoma.

Ferroptosis at the crossroads of tumor-host interactions, metastasis, and therapy response.

Monoubiquitination in Homeostasis and Cancer.

USP7 substrates identified by proteomics analysis reveal the specificity of USP7.

LIF-ting ferroptosis to improve liver cancer therapy

Glucocorticoid receptor regulates PD-L1 and MHC-I in pancreatic cancer cells to promote immune evasion and immunotherapy resistance.

A targetable LIFR-NF-κB-LCN2 axis controls liver tumorigenesis and vulnerability to ferroptosis.

Non-canonical function of DGCR8 in DNA double-strand break repair signaling and tumor radioresistance.

Collaborators (7)

Alexandre Gaspar-Maia

Di Zhao

Yutong Sun

Nathalie Droin

Hao Zhu

Linghua Wang

Shinichi Nakagawa

Make your next move.

Li Ma

Research Interests

Explore related searches

Contact this professor

About

Articles (20)

Sickle cell disease induces chromatin introversion and ferroptosis in CD8&lt;sup&gt;+&lt;/sup&gt; T cells to suppress anti-tumor immunity.

In vivo CRISPR activation screen identifies acyl-CoA-binding protein as a driver of bone metastasis.

Histone methyltransferase ASH1L primes metastases and metabolic reprogramming of macrophages in the bone niche.

Long noncoding RNA Malat1 protects against osteoporosis and bone metastasis

RNA Shielding of p65 Is Required to Potentiate Oncogenic Inflammation in TET2-Mutated Clonal Hematopoiesis.

Barriers and opportunities in pancreatic cancer immunotherapy

LIFR regulates cholesterol-driven bidirectional hepatocyte-neutrophil cross-talk to promote liver regeneration.

DePARylation is critical for S phase progression and cell survival

A milestone in epithelial–mesenchymal transition

Role of Glucose Metabolic Reprogramming in Breast Cancer Progression and Drug Resistance.

New Insights into YAP/TAZ-TEAD-Mediated Gene Regulation and Biological Processes in Cancer

ZRANB1 is an NBS1 deubiquitinase and a potential target to overcome radioresistance and PARP inhibitor resistance in triple-negative breast cancer.

Immunotherapeutic Approaches for Treating Hepatocellular Carcinoma.

Ferroptosis at the crossroads of tumor-host interactions, metastasis, and therapy response.

Monoubiquitination in Homeostasis and Cancer.

USP7 substrates identified by proteomics analysis reveal the specificity of USP7.

LIF-ting ferroptosis to improve liver cancer therapy

Glucocorticoid receptor regulates PD-L1 and MHC-I in pancreatic cancer cells to promote immune evasion and immunotherapy resistance.

A targetable LIFR-NF-κB-LCN2 axis controls liver tumorigenesis and vulnerability to ferroptosis.

Non-canonical function of DGCR8 in DNA double-strand break repair signaling and tumor radioresistance.

Collaborators (7)

Alexandre Gaspar-Maia

Di Zhao

Yutong Sun

Nathalie Droin

Hao Zhu

Linghua Wang

Shinichi Nakagawa

Make your next move.

Sickle cell disease induces chromatin introversion and ferroptosis in CD8<sup>+</sup> T cells to suppress anti-tumor immunity.