Linda Diehl

Prof.

University Medical Center Hamburg-Eppendorf

Germany

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Professor Linda Diehl is a faculty member at the University Medical Center Hamburg-Eppendorf in Germany. Her research interests include tumor and immune cell interactions, immune regulation mechanisms, and antigen presentation in various disease models. Recent publications explore topics such as myeloid-derived suppressor cells, tumor metastasis, and the immune response in lung cancer and autoimmune liver disease.

Articles (12)

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2024

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2024

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (1)

Gerrit Wolters-Eisfeld

University Medical Center Hamburg-Eppendorf

GERMANY

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Linda Diehl

Research Interests

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Articles (12)

CD44 expression on murine hepatic stellate cells promotes the induction of monocytic and polymorphonuclear myeloid-derived suppressor cells.

Editorial: Tumor and immune cell interactions in the formation of organ-specific metastasis.

Tumor cell integrin β4 and tumor stroma E-/P-selectin cooperatively regulate tumor growth in vivo.

The ADP-Ribosylation Factor 4d Restricts Regulatory T-Cell Induction via Control of IL-2 Availability.

Antigen Cross-Presentation by Murine Proximal Tubular Epithelial Cells Induces Cytotoxic and Inflammatory CD8<sup>+</sup> T Cells.

Smad7 Deficiency in Myeloid Cells Does Not Affect Liver Injury, Inflammation or Fibrosis after Chronic CCl4 Exposure in Mice.

Immune related endonucleases and GTPases are not associated with tumor response in patients with advanced non-small cell lung cancer treated with checkpoint inhibitors.

Comparison of Resistance Spectra after First and Second Line Osimertinib Treatment Detected by Liquid Biopsy.

Notch1 Deficiency Induces Tumor Cell Accumulation Inside the Bronchiolar Lumen and Increases TAZ Expression in an Autochthonous <i>Kras</i> <sup>LSL-G12V</sup> Driven Lung Cancer Mouse Model.

Antigen presentation, autoantibody production, and therapeutic targets in autoimmune liver disease.

Pioglitazone-Mediated Peroxisome Proliferator-Activated Receptor γ Activation Aggravates Murine Immune-Mediated Hepatitis.

TP53 co-mutations in EGFR mutated patients in NSCLC stage IV: A strong predictive factor of ORR, PFS and OS in EGFR mt+ NSCLC.

Collaborators (1)

Gerrit Wolters-Eisfeld

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