Lisa Rimsza

Consultant and Professor

Mayo Clinic Arizona

United States

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Lisa Rimsza is a Consultant and Professor at Mayo Clinic Arizona in the United States. Her research focuses on the molecular and cellular underpinnings of diffuse large B-cell lymphoma, as well as the clinical parameters relevant to mantle cell lymphoma. Recent publications encompass topics such as lysosomal inhibition, therapeutic strategies for various lymphoma subtypes, and the impact of storage conditions on biological samples.

Articles (10)

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2025

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2021

Collaborators (4)

Matthew Maurer

Assistant Professor of Biostatistics and Medicine

Mayo Clinic Rochester

UNITED STATES

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Alyssa Bouska

University of Nebraska Medical Center

UNITED STATES

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Christopher Thom

University of Pennsylvania

UNITED STATES

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Alina S. Gerrie

The University of British Columbia

CANADA

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Lisa Rimsza

Research Interests

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Articles (10)

Evaluation of Etiologic Heterogeneity for Risk of Diffuse Large B-Cell Lymphoma Subtype Defined by the Cell of Origin

Evaluation of clinical parameters and biomarkers in older, untreated mantle cell lymphoma patients receiving bendamustine–rituximab

CDK8/19 inhibition triggers a switch from mitosis to endomitosis in cord blood megakaryocytes

Alport syndrome: A puzzling case of thrombocytopenia and giant unusual platelets in blood smear

“Quadruple-hit” primary testicular diffuse large B-cell lymphoma with MYD88 L265P mutation, IGH::MYC, and IRF4- and BCL6-rearrangements

Transformation of FL into DLBCL with a PMBL gene expression signature

Bendamustine or high-dose cytarabine-based induction with rituximab in transplant-eligible mantle cell lymphoma.

Assessment of 2-Year Storage Conditions on Protein, RNA, and DNA in Unstained Human Tissue Sections, Including a Novel Multiplex Digital Gene Expression Profiling Method with Implications for Biobanking

Mediastinal B-cell lymphoma with MYC, BCL2, and BCL6 rearrangements

EBV-positive HIV-associated diffuse large B cell lymphomas are characterized by JAK/STAT (STAT3) pathway mutations and unique clinicopathologic features.

Collaborators (4)

Matthew Maurer

Alyssa Bouska

Christopher Thom

Alina S. Gerrie

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