Lisa Wiesmüller

Professor

Ulm University

Germany

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About

Professor Lisa Wiesmüller is a faculty member at Ulm University in Germany. Her research focuses on cancer biology, particularly the roles of p53 isoforms, DNA damage responses, and the efficacy of anti-cancer agents. Recent articles include investigations into ctDNA in breast cancer, the synergy of polymerase inhibitors, and the impact of cellular conditions on DNA damage repair mechanisms.

Articles (14)

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2025

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2025

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (8)

Peter Bouwman

Assistant professor

NETHERLANDS

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Meinrad Beer

Ulm University Medical Center

GERMANY

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Ruth, PD Dr, Hemmersbach

Deputy Head

German Aerospace Center

GERMANY

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Ralph Scully

Professor

Beth Israel Deaconess Medical Center

UNITED STATES

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Helmut Pospiech

Head of the Laboratory

University Hospital Düsseldorf

GERMANY

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Diana M Eccles

University of Southampton

UNITED KINGDOM

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Melanie Rall-Scharpf

Ulm University

GERMANY

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Marjanka Kathelina Schmidt

Professor of genetic epidemiology of (breast) cancer

Utrecht University

NETHERLANDS

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Lisa Wiesmüller

Research Interests

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Articles (14)

Is Radiographic Aftercare Obsolete? How Testing Positive for ctDNA Can Be a Precedent for Late Relapse, Even in Low-Risk Hormone-Receptor-Positive Breast Cancer

Versatile enhancement of the killing potential of anti-cancer agents achieved by peptide mimetics of the PCNA interface towards specialized DNA polymerases

Canonical and non-canonical functions of p53 isoforms: potentiating the complexity of tumor development and therapy resistance

The levels of p53 govern the hierarchy of DNA damage tolerance pathway usage

Simulating Space Conditions Evokes Different DNA Damage Responses in Immature and Mature Cells of the Human Hematopoietic System

Polymerase ζ Is Involved in Mitochondrial DNA Maintenance Processes in Concert with APE1 Activity

Combinations of ATR, Chk1 and Wee1 Inhibitors with Olaparib Are Active in Olaparib Resistant <i>Brca1</i> Proficient and Deficient Murine Ovarian Cells

Circulating Tumor Cells in Breast Cancer Patients: A Balancing Act between Stemness, EMT Features and DNA Damage Responses

p53 isoforms differentially impact on the POLι dependent DNA damage tolerance pathway

Impact of the interplay between stemness features, p53 and pol iota on replication pathway choices

Dual PARP and RAD51 Inhibitory Drug Conjugates Show Synergistic and Selective Effects on Breast Cancer Cells

Multiple biochemical properties of the p53 molecule contribute to activation of polymerase iota-dependent DNA damage tolerance

Variants of uncertain clinical significance in hereditary breast and ovarian cancer genes: best practices in functional analysis for clinical annotation

53BP1 Accumulation in Circulating Tumor Cells Identifies Chemotherapy-Responsive Metastatic Breast Cancer Patients

Collaborators (8)

Peter Bouwman

Meinrad Beer

Ruth, PD Dr, Hemmersbach

Ralph Scully

Helmut Pospiech

Diana M Eccles

Melanie Rall-Scharpf

Marjanka Kathelina Schmidt

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