Luca Rampoldi

Head of Research Unit

Ospedale San Raffaele

Italy

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About

Dr. Luca Rampoldi is the Head of Research Unit at Ospedale San Raffaele in Italy. His research primarily focuses on kidney diseases, particularly in relation to uromodulin and autosomal dominant tubulointerstitial kidney disease. Recent publications highlight areas such as mitochondrial dysfunction, ER stress, and the genetic and clinical predictors affecting kidney health.

Articles (11)

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2025

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2023

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2023

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2021

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2021

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2020

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2020

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2020

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2020

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2020

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2020

Collaborators (8)

Delyth Graham

Senior Lecturer

University of Glasgow

UNITED KINGDOM

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Sheon Samji

University of Glasgow

UNITED KINGDOM

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Jakub Sikora

assistant professor

Charles University, First Faculty of Medicine

CZECH REPUBLIC

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Jan Zivny

Charles University in Prague

CZECH REPUBLIC

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Guglielmo Schiano

University of Zurich

SWITZERLAND

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Joaquim Calado

Universidade de Aveiro

PORTUGAL

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Federico Alberici

University of Brescia

ITALY

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Agnieszka Łaszkiewicz

Hirszfeld Institute of Immunology and Experimental Therapy, Polish Academy of Sciences

POLAND

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Luca Rampoldi

Research Interests

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Articles (11)

Early renal response to long-term salt loading: mitochondrial dysfunction, ER stress, and uromodulin accumulation in the kidney medulla

Allelic effects on uromodulin aggregates drive autosomal dominant tubulointerstitial kidney disease

Leader peptide or pro-segment mutants of renin are misrouted to mitochondria in autosomal dominant tubulointerstitial kidney disease

Mechanistic interactions of uromodulin with the thick ascending limb: perspectives in physiology and hypertension

Uromodulin: Roles in Health and Disease.

Genetic and Clinical Predictors of Age of ESKD in Individuals With Autosomal Dominant Tubulointerstitial Kidney Disease Due to UMOD Mutations

An international cohort study of autosomal dominant tubulointerstitial kidney disease due to REN mutations identifies distinct clinical subtypes

Variable Expressivity of HNF1B Nephropathy, From Renal Cysts and Diabetes to Medullary Sponge Kidney Through Tubulo-interstitial Kidney Disease

Chemically based transmissible ER stress protocols are unsuitable to study cell-to-cell UPR transmission.

The Interaction of the Tumor Suppressor FAM46C with p62 and FNDC3 Proteins Integrates Protein and Secretory Homeostasis.

Clinical and genetic spectra of autosomal dominant tubulointerstitial kidney disease due to mutations in UMOD and MUC1.

Collaborators (8)

Delyth Graham

Sheon Samji

Jakub Sikora

Jan Zivny

Guglielmo Schiano

Joaquim Calado

Federico Alberici

Agnieszka Łaszkiewicz

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