Luciana Berod

Germany

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Articles (15)

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2025

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2025

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2024

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2021

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2020

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2020

Collaborators (7)

Ayesha Dhillon-LaBrooy

Universitätsmedizin der Johannes Gutenberg-Universität Mainz

GERMANY

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Tim Sparwasser

Director/ Professor

University Medical Center of the Johannes Gutenberg University Mainz

GERMANY

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Diana Moreira

Trinity College Dublin

IRELAND

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Manfred B. Lutz

Associate Professor

University of Würzburg

GERMANY

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David Allan

National Heart Lung and Blood Institute

UNITED STATES

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Venetia Bigley

MRC Senior Clinical Fellow, Clinical Senior Lecturer in Haematology

Newcastle University

UNITED KINGDOM

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Anne B. Krug

Ludwig-Maximilians-Universität München

GERMANY

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Luciana Berod

Research Interests

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Articles (15)

ACC1 is a dual metabolic-epigenetic regulator of Treg stability and immune tolerance

Metabolic reprogramming of interleukin-17-producing γδ T cells promotes ACC1-mediated de novo lipogenesis under psoriatic conditions

Inhibition of Mitochondrial Translation Ameliorates Imiquimod-Induced Psoriasis-Like Skin Inflammation by Targeting Vy4D y8 T Cells

Regulation of DC metabolism by nitric oxide in murine GM-CSF cultures

Inhibition of Mitochondrial Translation Ameliorates Imiquimod-Induced Psoriasis-Like Skin Inflammation by Targeting Vγ4+ γδ T Cells

Targeting ACC1 in T cells ameliorates psoriatic skin inflammation

Guidelines for mouse and human DC functional assays

Regulation of DC metabolism by nitric oxide in murine GM-CSF cultures

Targeting ACC1 in T cells ameliorates psoriatic skin inflammation

Guidelines for mouse and human DC generation

CD4<sup>+</sup> T-cell differentiation and function: Unifying glycolysis, fatty acid oxidation, polyamines NAD mitochondria

Dendritic cell metabolism: moving beyond in vitro-culture-generated paradigms

Ribosome-Targeting Antibiotics Impair T Cell Effector Function and Ameliorate Autoimmunity by Blocking Mitochondrial Protein Synthesis

Targeting cellular fatty acid synthesis limits T helper and innate lymphoid cell function during intestinal inflammation and infection.

The Absence of HIF-1α Increases Susceptibility to Leishmania donovani Infection via Activation of BNIP3/mTOR/SREBP-1c Axis

Collaborators (7)

Ayesha Dhillon-LaBrooy

Tim Sparwasser

Diana Moreira

Manfred B. Lutz

David Allan

Venetia Bigley

Anne B. Krug

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