M. Lienhard Schmitz

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Full Professor

University of Giessen

Germany

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Dr. M. Lienhard Schmitz is a Full Professor at the University of Giessen, Germany. His research focuses on cancer biology, specifically the metabolic targeting of cancer cells, NF-κB signaling, and mitochondrial metabolism. He has contributed to understanding the epigenetic regulation of genes in cancer and the cardiac response to pressure overload. His recent publications reflect his interests in the cellular and molecular mechanisms underlying disease processes.

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Articles (21)

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Collaborators (5)

Eva Böttcher-Friebertshäuser

Philipps-Universität Marburg

GERMANY

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Ingrid Fleming

Goethe University Frankfurt

GERMANY

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John Ziebuhr

Professor

Justus Liebig University

GERMANY

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Sven Zukunft

Medizinische Fakultät Carl Gustav Carus, Technische Universität Dresden

GERMANY

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Johannes Graumann

Professor of Translational Proteomics

Philipps-Universität Marburg

GERMANY

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M. Lienhard Schmitz

Research Interests

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Contact this professor

About

Recent Grants

Articles (21)

Synergistic targeting of cancer cells through simultaneous inhibition of key metabolic enzymes

Insights from human NF-κB knockouts

SIAH3 is frequently epigenetically silenced in cancer and regulates mitochondrial metabolism

The proximity-based protein interactome and regulatory logics of the transcription factor p65 NF-κB/RELA

Distinct negative-sense RNA viruses induce a common set of transcripts encoding proteins forming an extensive network

Comparison of the stage-dependent mitochondrial changes in response to pressure overload between the diseased right and left ventricle in the rat

The Aurora B-controlled PP1/RepoMan complex determines the spatial and temporal distribution of mitotic H2B S6 phosphorylation

A reductionist perspective on HIF-1α's role in cell proliferation under non-hypoxic conditions

Open questions in the NF-κB field

A common gene signature of the right ventricle in failing rat and human hearts

A transient increase of HIF-1α during the G1 phase (G1-HIF) ensures cell survival under nutritional stress

Introducing the Special Issue on "Emerging Concepts & Non-Canonical Pathways in Cellular Signaling"

Phosphorylation of the PA subunit of influenza polymerase at Y393 prevents binding of the 5′-termini of RNA and polymerase function

Release of STK24/25 suppression on MEKK3 signaling in endothelial cells confers cerebral cavernous malformation

Phosphorylation of Influenza A Virus Matrix Protein 1 at Threonine 108 Controls Its Multimerization State and Functional Association with the STRIPAK Complex

Testing the Effect of Histone Acetyltransferases on Local Chromatin Compaction.

Understanding and Therapeutic Targeting of the p53 Network

Reply to: The stress-inducible ER chaperone GRP78/BiP is upregulated during SARS-CoV-2 infection and acts as a pro-viral protein

Post-Transcriptional Control of mRNA Metabolism and Protein Secretion: The Third Level of Regulation within the NF-κB System

Therapeutic induction of Bcl2-associated athanogene 3-mediated autophagy in idiopathic pulmonary fibrosis.

Comparative kinase activity profiling of pathogenic influenza A viruses reveals new anti- and pro-viral protein kinases

Collaborators (5)

Eva Böttcher-Friebertshäuser

Ingrid Fleming

John Ziebuhr

Sven Zukunft

Johannes Graumann

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