Maciej Lech

Prof./Habil./PhD

LMU Klinikum

Germany

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Professor Maciej Lech is a Habilitated Doctor of Philosophy at LMU Klinikum in Germany. His research primarily focuses on kidney disease, inflammatory responses, and macrophage biology, as evidenced by his recent publications on podocyte injury and the mechanisms underlying glomerulonephritis. He is actively exploring novel therapeutic targets for kidney-related ailments and the role of immune regulation in diseases such as lupus nephritis.

Articles (12)

Year:

2025

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2022

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2022

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2021

Collaborators (9)

Anna Golda

Jagiellonian University

POLAND

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Stefanie Steiger

University Hospital

GERMANY

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Ekaterina von Rauchhaupt

Heidelberg University

GERMANY

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Jerzy Kotlinowski

Jagiellonian University

POLAND

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Joanna Koziel

POLAND

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Hans-Joachim Anders

GERMANY

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Manikandan Subramanian

UNITED KINGDOM

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Andreas Margraf

University Hospital Münster

GERMANY

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Jan Potempa

Prof. dr hab.

Uniwersytet Jagielloński w Krakowie

POLAND

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Maciej Lech

Research Interests

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Articles (12)

Podocyte A20/TNFAIP3 Controls Glomerulonephritis Severity via the Regulation of Inflammatory Responses and Effects on the Cytoskeleton

GDF-15 Suppresses Puromycin Aminonucleoside-Induced Podocyte Injury by Reducing Endoplasmic Reticulum Stress and Glomerular Inflammation

Uremic Toxin Indoxyl Sulfate Promotes Macrophage-Associated Low-Grade Inflammation and Epithelial Cell Senescence

Promising novel therapeutic targets for kidney disease: Emphasis on kidney-specific proteins

Macrophage-Specific MCPIP1/Regnase-1 Attenuates Kidney Ischemia-Reperfusion Injury by Shaping the Local Inflammatory Response and Tissue Regeneration

A randomized prospective cross over study on the effects of medium cut-off membranes on T cellular and serologic immune phenotypes in hemodialysis

Editorial: Negative Regulators of Innate Immunity and Their Role in Host Responses to Injury and Infection

GDF15 Suppresses Lymphoproliferation and Humoral Autoimmunity in a Murine Model of Systemic Lupus Erythematosus

Hypercholesterolemia promotes autoantibody production and a lupus-like pathology via decreased DNase-mediated clearance of DNA

Lupus Nephritis: Current Perspectives and Moving Forward

MCP-Induced Protein 1 Participates in Macrophage-Dependent Endotoxin Tolerance

Murine myeloid cell MCPIP1 suppresses autoimmunity by regulating B-cell expansion and differentiation

Collaborators (9)

Anna Golda

Stefanie Steiger

Ekaterina von Rauchhaupt

Jerzy Kotlinowski

Joanna Koziel

Hans-Joachim Anders

Manikandan Subramanian

Andreas Margraf

Jan Potempa

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