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Magdalena Koczkowska

Assistant Professor

Medical University of Gdansk

Poland

Research Interests

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Articles (10)

Year:

2025

Year:

2025

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2025

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2025

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2024

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2024

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2024

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2023

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2023

Year:

2023

Collaborators (9)

Urszula Ławrynowicz

Medical University of Gdansk

POLAND

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Anna Marcinkowska

Assisstant Professor

Medical University of Gdansk

POLAND

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Katarzyna Czerwiec

Reserch assistant

Medical University of Gdansk

POLAND

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Małgorzata Zawrzykraj

Assistant professor

Medical University of Gdansk

POLAND

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Izabella Kuźmiuk-Glembin

MD, PhD

Medical University of Gdansk

POLAND

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Aneta Skoniecka

Assistant Professor

Medical University of Gdansk

POLAND

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Deeann Wallis

The University of Alabama

UNITED STATES

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Pawel Winklewski

Professor

Gdański Uniwersytet Medyczny

POLAND

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Bartosz Wasąg

Head of Department

Medical University of Gdansk

POLAND

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Magdalena Koczkowska

Research Interests

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Articles (10)

Differences in the neuropsychological profiles of high-functioning TSC adults with and without epilepsy

Alginate formulations with high loads of zebularine and retinoic acid promote tissue growth and innervation and induce extensive epigenetic repatterning

Comprehensive genetic screening of 70 severe adolescent idiopathic scoliosis probands reveals novel pathogenic variants and syndromic associations

Identifying differentiation markers between dermal fibroblasts and adipose-derived mesenchymal stromal cells (AD-MSCs) in human visceral and subcutaneous tissues using single-cell transcriptomics

Genotype–Phenotype Correlation in Neurofibromatosis Type 1: Evidence for a Mild Phenotype Associated with Splicing Variants Leading to In-Frame Skipping of <i>NF1</i> Exon 24 [19a]

Adipose-derived mesenchymal stromal cells in clinical trials: Insights from single-cell studies

Beyond Tumors: Reduced survival linked to pathogenic PIK3CA and TP53 post-zygotic variants in uninvolved mammary tissue of breast cancer patients with recurrent disease

Analysis of BRCA1 and BRCA2 alternative splicing in predisposition to ovarian cancer.

Analysis of 200 unrelated individuals with a constitutional <i>NF1</i> deep intronic pathogenic variant reveals that variants flanking the alternatively spliced <i>NF1</i> exon 31 [23a] cause a classical neurofibromatosis type 1 phenotype while altering predominantly <i>NF1</i> isoform type II

Size matters: the impact of nucleus size on results from spatial transcriptomics

Collaborators (9)

Urszula Ławrynowicz

Anna Marcinkowska

Katarzyna Czerwiec

Małgorzata Zawrzykraj

Izabella Kuźmiuk-Glembin

Aneta Skoniecka

Deeann Wallis

Pawel Winklewski

Bartosz Wasąg

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