Mai Kishimoto

Assistant professor

Osaka University

Japan

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About

Mai Kishimoto is an Assistant Professor at Osaka Metropolitan University, Japan. Her research focuses on virology, with recent articles addressing various aspects of SARS-CoV-2, including its protease inhibitors, variants, and pathogenicity. Additionally, she has explored orthoreoviruses from fruit bats and the unique promoter structures within the Bunyavirales family.

Articles (10)

Year:

2023

Year:

2022

Year:

2022

Year:

2022

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2022

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2022

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2022

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2021

Year:

2021

Year:

2021

Collaborators (10)

Michihito Sasaki

JAPAN

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Hirofumi Sawa

Hokkaido University

JAPAN

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Kumiko Morimatsu

Associate Professor

JAPAN

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Takamasa Ueno

Professor

Kumamoto University

JAPAN

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Shinya Tanaka

Hokkaido University

JAPAN

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Kanako Kimura

Assistant Professor

Kyoto University

JAPAN

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Kotaro Shirakawa

講師 / Senior Lecturer/ Junior Associate Professor

Kyoto University

JAPAN

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Terumasa Ikeda

Associate Professor

Kumamoto University

JAPAN

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Jumpei Ito

University of Tokyo

JAPAN

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Satoshi Taniguchi

University of Tokyo

JAPAN

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Mai Kishimoto

Research Interests

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About

Articles (10)

S-217622, a SARS-CoV-2 main protease inhibitor, decreases viral load and ameliorates COVID-19 severity in hamsters

Attenuated fusogenicity and pathogenicity of SARS-CoV-2 Omicron variant

Virological characteristics of the SARS-CoV-2 Omicron BA.2 spike

Serological characterization of lineage II insect-specific flaviviruses compared with pathogenic mosquito-borne flaviviruses

Isolation and characterization of an orthoreovirus from Indonesian fruit bats

Virological characteristics of the SARS-CoV-2 Omicron BA.2 subvariants, including BA.4 and BA.5

A comprehensive list of the Bunyavirales replication promoters reveals a unique promoter structure in Nairoviridae differing from other virus families

Air-liquid interphase culture confers SARS-CoV-2 susceptibility to A549 alveolar epithelial cells

SARS-CoV-2 Bearing a Mutation at the S1/S2 Cleavage Site Exhibits Attenuated Virulence and Confers Protective Immunity

Host serine proteases TMPRSS2 and TMPRSS11D mediate proteolytic activation and trypsin-independent infection in group A rotaviruses.

Collaborators (10)

Michihito Sasaki

Hirofumi Sawa

Kumiko Morimatsu

Takamasa Ueno

Shinya Tanaka

Kanako Kimura

Kotaro Shirakawa

Terumasa Ikeda

Jumpei Ito

Satoshi Taniguchi

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